Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure. - Wikidata - wikimedia - TriplyDB

Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

http://www.wikidata.org/entity/Q36896507

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The Role of Mitochondrial Functional Proteins in ROS Production in Ischemic Heart Diseases Role of β-hydroxybutyrate, its polymer poly-β-hydroxybutyrate and inorganic polyphosphate in mammalian health and disease Inhaled matters of the heart Is treatment with trimetazidine beneficial in patients with chronic heart failure?Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate.Caffeic acid ethanolamide prevents cardiac dysfunction through sirtuin dependent cardiac bioenergetics preservation.Cytosolic and nuclear calcium signaling in atrial myocytes: IP3-mediated calcium release and the role of mitochondria.Combined Inhibition of the Renin-Angiotensin System and Neprilysin Positively Influences Complex Mitochondrial Adaptations in Progressive Experimental Heart Failure Trimetazidine does not alter metabolic substrate oxidation in cardiac mitochondria of target patient population.Chlorine-induced cardiopulmonary injury BNC Protects H9c2 Cardiomyoblasts from H 2 O 2 -Induced Oxidative Injury through ERK1/2 Signaling Pathway.Effect of reproductive ageing on pregnant mouse uterus and cervix.Mitochondrial dynamics in the heart as a novel therapeutic target for cardioprotection.Mitochondrial oxidative metabolism and uncoupling proteins in the failing heart.The reduced myofilament responsiveness to calcium contributes to the negative force-frequency relationship in rat cardiomyocytes: role of reactive oxygen species and p-38 map kinase.Dyssynchronous calcium removal in heart failure-induced atrial remodeling.Salicylic Acid-Dependent Plant Stress Signaling via Mitochondrial Succinate Dehydrogenase.Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β-adrenergic stimulation.Cardioprotection by curcumin post-treatment in rats with established chronic kidney disease.Effects of trimetazidine on mitochondrial respiratory function, biosynthesis, and fission/fusion in rats with acute myocardial ischemia.Trimetazidine restores the positive adaptation to exercise training by mitigating statin-induced skeletal muscle injury.Mitochondrial Bioenergetics During Ischemia and Reperfusion.Study of respiratory chain dysfunction in heart disease.Cardiac-specific Conditional Knockout of the 18-kDa Mitochondrial Translocator Protein Protects from Pressure Overload Induced Heart Failure

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Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

http://www.wikidata.org/entity/Q36896507

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

@en

type

label

Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

@en

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Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

@en

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J.YJMCC.2013.01.016

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Journal of Molecular and Cellular Cardiology

P1476

Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.

@en

P2093

Elena N Dedkova

http://www.w3.org/2001/XMLSchema#string

Lea K Seidlmayer

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Lothar A Blatter

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P2860

Nitric oxide: physiology, pathophysiology, and pharmacology

Mitochondrial permeability transition in cardiac cell injury and death

The antianginal drug trimetazidine shifts cardiac energy metabolism from fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-ketoacyl coenzyme A thiolase

The mitochondrial permeability transition pore and its role in cell death

Mitochondrial calcium uptake stimulates nitric oxide production in mitochondria of bovine vascular endothelial cells.

A fluorescently labeled intestinal fatty acid binding protein. Interactions with fatty acids and its use in monitoring free fatty acids.

Modulation of mitochondrial Ca2+ by nitric oxide in cultured bovine vascular endothelial cells.

Opening of mitochondrial permeability transition pore induces hypercontracture in Ca2+ overloaded cardiac myocytes.

Characteristics and function of cardiac mitochondrial nitric oxide synthase.

Oxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load.

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41-54

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scholarly article

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10.1016/J.YJMCC.2013.01.016

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59

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2013-02-04T00:00:00Z

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