Generation of glucocorticoid-responsive Moloney murine leukemia virus by insertion of regulatory sequences from murine mammary tumor virus into the long terminal repeat.
about
Tandemization of a subregion of the enhancer sequences from SRS 19-6 murine leukemia virus associated with T-lymphoid but not other leukemias.A chimeric Ty3/Moloney murine leukemia virus integrase protein is active in vivo.Glucocorticoid regulation of murine leukemia virus transcription elements is specified by determinants within the viral enhancer region.A pancreas specificity results from the combination of polyomavirus and Moloney murine leukemia virus enhancer.Transcriptional repression of a hormone-responsive promoterHepatitis B virus DNA contains a glucocorticoid-responsive element.A redundant nuclear protein binding site contributes to negative regulation of the mouse mammary tumor virus long terminal repeat.Substitution of murine transthyretin (prealbumin) regulatory sequences into the Moloney murine leukemia virus long terminal repeat yields infectious virus with altered biological propertiesDeletion of a GC-rich region flanking the enhancer element within the long terminal repeat sequences alters the disease specificity of Moloney murine leukemia virus.Alignment of U3 region sequences of mammalian type C viruses: identification of highly conserved motifs and implications for enhancer designNegative regulatory element associated with potentially functional promoter and enhancer elements in the long terminal repeats of endogenous murine leukemia virus-related proviral sequences.Chromatin structure of recombinant Moloney murine leukemia virus proviral DNAs that contain tax-responsive sequences from human T-cell lymphotropic virus type II in the presence and absence of tax.Six distinct nuclear factors interact with the 75-base-pair repeat of the Moloney murine leukemia virus enhancerNegative regulation of transcription in vitro by a glucocorticoid response element is mediated by a trans-acting factor.Addition of substitution of simian virus 40 enhancer sequences into the Moloney murine leukemia virus (M-MuLV) long terminal repeat yields infectious M-MuLV with altered biological propertiesRearrangements and insertions in the Moloney murine leukemia virus long terminal repeat alter biological properties in vivo and in vitro.Generation of infectious Moloney murine leukemia viruses with deletions in the U3 portion of the long terminal repeatTwo different factors act separately or together to specify functionally distinct activities at a single transcriptional enhancer.Construction and characterization of a hybrid mouse mammary tumor virus/murine leukemia virus-based retroviral vector.Regulatory elements within the murine leukemia virus enhancer regions mediate glucocorticoid responsiveness.Sequences responsible for erythroid and lymphoid leukemia in the long terminal repeats of Friend-mink cell focus-forming and Moloney murine leukemia viruses.
P2860
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P2860
Generation of glucocorticoid-responsive Moloney murine leukemia virus by insertion of regulatory sequences from murine mammary tumor virus into the long terminal repeat.
description
1985 nî lūn-bûn
@nan
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
1985年论文
@zh
1985年论文
@zh-cn
name
Generation of glucocorticoid-r ...... into the long terminal repeat.
@en
type
label
Generation of glucocorticoid-r ...... into the long terminal repeat.
@en
prefLabel
Generation of glucocorticoid-r ...... into the long terminal repeat.
@en
P2860
P1433
P1476
Generation of glucocorticoid-r ...... into the long terminal repeat
@en
P2093
P2860
P304
P407
P577
1985-04-01T00:00:00Z