BHK cells expressing Sindbis virus-induced homologous interference allow the translation of nonstructural genes of superinfecting virus.
about
Dual mechanisms of pestiviral superinfection exclusion at entry and RNA replication.Superinfection Exclusion in Cells Infected with Hepatitis C VirusAnalysis of Hepatitis C Virus Superinfection Exclusion by Using Novel Fluorochrome Gene-Tagged Viral GenomesSuperinfection exclusion of alphaviruses in three mosquito cell lines persistently infected with Sindbis virusTransdominant Inhibition of Bovine Viral Diarrhea Virus EntryMutations which alter the level or structure of nsP4 can affect the efficiency of Sindbis virus replication in a host-dependent mannerExclusion of superinfecting homologous virus by Sindbis virus-infected Aedes albopictus (mosquito) cellsEffect of actinomycin D and cycloheximide on replication of Sindbis virus in Aedes albopictus (mosquito) cellsThe alphaviruses: gene expression, replication, and evolutionPersistent Hz-1 virus infection in insect cells: evidence for insertion of viral DNA into host chromosomes and viral infection in a latent status.A novel mode of poxvirus superinfection exclusion that prevents fusion of the lipid bilayers of viral and cellular membranesWhy do RNA viruses recombine?A viral protein mediates superinfection exclusion at the whole-organism level but is not required for exclusion at the cellular level.Alphavirus minus-strand synthesis and persistence in mouse embryo fibroblasts derived from mice lacking RNase L and protein kinase R.Eilat virus induces both homologous and heterologous interference.A Drosophila toolkit for the visualization and quantification of viral replication launched from transgenic genomes.Superinfection exclusion is an active virus-controlled function that requires a specific viral proteinSuperinfection-induced apoptosis and its correlation with the reduction of viral progeny in cells persistently infected with Hz-1 baculovirusDeveloping an understanding of cross-protection by Citrus tristeza virus.Rescue of Sindbis virus-specific RNA replication and transcription by using a vaccinia virus recombinant.Transgenic mice expressing the nucleoprotein of Borna disease virus in either neurons or astrocytes: decreased susceptibility to homotypic infection and disease.Evasion of superinfection exclusion and elimination of primary viral RNA by an adapted strain of hepatitis C virus.Properties of non-structural protein 1 of Semliki Forest virus and its interference with virus replicationThe Coat Protein and NIa Protease of Two Potyviridae Family Members Independently Confer Superinfection Exclusion.Selective virus resistance conferred by expression of Borna disease virus nucleocapsid components.Infection with strains of Citrus tristeza virus does not exclude superinfection by other strains of the virus.Superinfection Exclusion between Two High-Risk Human Papillomavirus (HPV) Types During a Co-Infection.
P2860
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P2860
BHK cells expressing Sindbis virus-induced homologous interference allow the translation of nonstructural genes of superinfecting virus.
description
1985 nî lūn-bûn
@nan
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
1985年论文
@zh
1985年论文
@zh-cn
name
BHK cells expressing Sindbis v ...... genes of superinfecting virus.
@en
type
label
BHK cells expressing Sindbis v ...... genes of superinfecting virus.
@en
prefLabel
BHK cells expressing Sindbis v ...... genes of superinfecting virus.
@en
P2860
P1433
P1476
BHK cells expressing Sindbis v ...... genes of superinfecting virus.
@en
P2093
P2860
P304
P407
P577
1985-05-01T00:00:00Z