Thiamine-dependent processes and treatment strategies in neurodegeneration.
about
Role of dietary protein and thiamine intakes on cognitive function in healthy older people: a systematic reviewProstatic acid phosphatase is required for the antinociceptive effects of thiamine and benfotiamineAlcohol and thiamine deficiency trigger differential mitochondrial transition pore opening mediating cellular deathInactivation of the DNA repair genes mutS, mutL or the anti-recombination gene mutS2 leads to activation of vitamin B1 biosynthesis genes.Effect of chronic kidney disease on the expression of thiamin and folic acid transportersNew considerations on the neuromodulatory role of thiamine.Mitochondrial protection attenuates inflammation-induced impairment of neurogenesis in vitro and in vivo.Neuronal MCP-1 mediates microglia recruitment and neurodegeneration induced by the mild impairment of oxidative metabolism.Mitochondrial energetics and therapeutics.Change in psychiatric symptomatology after benfotiamine treatment in males is related to lifetime alcoholism severity.Dysautonomia in autism spectrum disorder: case reports of a family with review of the literatureMeasurement of Blood Thiamine Metabolites for Alzheimer's Disease Diagnosis.High-dose thiamine improves the symptoms of fibromyalgiaHigh-dose thiamine improves the symptoms of Friedreich's ataxiaHigh-dose thiamine as initial treatment for Parkinson's diseaseThiamine deficiency induces oxidative stress and exacerbates the plaque pathology in Alzheimer's mouse model.Focal thalamic degeneration from ethanol and thiamine deficiency is associated with neuroimmune gene induction, microglial activation, and lack of monocarboxylic acid transporters.Diet, nutrients and metabolism: cogs in the wheel driving Alzheimer's disease pathology?Thiamine Deficiency and Neurodegeneration: the Interplay Among Oxidative Stress, Endoplasmic Reticulum Stress, and Autophagy.Correlation of thiamine metabolite levels with cognitive function in the non-demented elderly.Powerful beneficial effects of benfotiamine on cognitive impairment and beta-amyloid deposition in amyloid precursor protein/presenilin-1 transgenic mice.Membrane Aging as the Real Culprit of Alzheimer's Disease: Modification of a Hypothesis.Exposure to pyrithiamine increases β-amyloid accumulation, Tau hyperphosphorylation, and glycogen synthase kinase-3 activity in the brain.Pork and Chicken Meals Similarly Impact on Cognitive Function and Strength in Community-Living Older Adults: A Pilot Study.Long-term treatment with thiamine as possible medical therapy for Friedreich ataxia.
P2860
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P2860
Thiamine-dependent processes and treatment strategies in neurodegeneration.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Thiamine-dependent processes and treatment strategies in neurodegeneration.
@en
type
label
Thiamine-dependent processes and treatment strategies in neurodegeneration.
@en
prefLabel
Thiamine-dependent processes and treatment strategies in neurodegeneration.
@en
P356
P1476
Thiamine-dependent processes and treatment strategies in neurodegeneration.
@en
P2093
Gary E Gibson
John P Blass
P304
P356
10.1089/ARS.2007.1766
P577
2007-10-01T00:00:00Z