Prothymosin α overexpression contributes to the development of pulmonary emphysema.
about
Chemotherapeutics-induced Oct4 expression contributes to drug resistance and tumor recurrence in bladder cancer.Dysregulated miR-34a-SIRT1-acetyl p65 axis is a potential mediator of immune activation in the colon during chronic simian immunodeficiency virus infection of rhesus macaques.Subcellular dissemination of prothymosin alpha at normal physiology: immunohistochemical vis-a-vis western blotting perspectiveEtoposide enhances antitumor efficacy of MDR1-driven oncolytic adenovirus through autoupregulation of the MDR1 promoter activity.Loss of nuclear prothymosin-α expression is associated with disease progression in human superficial bladder cancer.Prothymosin alpha-deficiency enhances anxiety-like behaviors and impairs learning/memory functions and neurogenesis.
P2860
Prothymosin α overexpression contributes to the development of pulmonary emphysema.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Prothymosin α overexpression contributes to the development of pulmonary emphysema.
@en
type
label
Prothymosin α overexpression contributes to the development of pulmonary emphysema.
@en
prefLabel
Prothymosin α overexpression contributes to the development of pulmonary emphysema.
@en
P2093
P2860
P356
P1476
Prothymosin α overexpression contributes to the development of pulmonary emphysema.
@en
P2093
Ai-Li Shiau
Bing-Hua Su
Chao-Liang Wu
Gia-Shing Shieh
Kuo-Jung Li
Te-Hsin Yen
Ya-Chieh Shiang
Yau-Lin Tseng
Yi-Cheng Chen
P2860
P2888
P356
10.1038/NCOMMS2906
P407
P577
2013-01-01T00:00:00Z