A new common integration region (int-3) for mouse mammary tumor virus on mouse chromosome 17.
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Endogenous MMTV proviruses induce susceptibility to both viral and bacterial pathogensIdentification of a common viral integration region in Cas-Br-E murine leukemia virus-induced non-T-, non-B-cell lymphomasGlucocorticoid and growth factor synergism requirement for Notch4 chromatin domain activationTyrosine kinase signalling in breast cancer: fibroblast growth factors and their receptorsTargeting the Notch signaling pathway in cancer therapeuticsFrom fly wings to targeted cancer therapies: a centennial for notch signalingIntracisternal type A particle-mediated activation of the Notch4/int3 gene in a mouse mammary tumor: generation of truncated Notch4/int3 mRNAs by retroviral splicing events.A new common integration site, Int7, for the mouse mammary tumor virus in mouse mammary tumors identifies a gene whose product has furin-like and thrombospondin-like sequencesPro-oncogenic and anti-oncogenic pathways: opportunities and challenges of cancer therapy.Induction of cyclin D1 transcription and CDK2 activity by Notch(ic): implication for cell cycle disruption in transformation by Notch(ic).Notch and the awesome power of genetics.The mouse homolog of the hst/k-FGF gene is adjacent to int-2 and is activated by proviral insertion in some virally induced mammary tumors.Sequences within the gag gene of mouse mammary tumor virus needed for mammary gland cell transformation.Oncogenic and tumor suppressor functions of Notch in cancer: it's NOTCH what you think.Hierarchical phosphorylation within the ankyrin repeat domain defines a phosphoregulatory loop that regulates Notch transcriptional activityCooperation between structural elements in hormono-regulated transcription from the mouse mammary tumor virus promoter.Targeted integration of adeno-associated virus (AAV) into human chromosome 19.Notch-induced mammary tumorigenesis does not involve the lobule-limited epithelial progenitorHeterogeneity of breast cancer stem cells as evidenced with Notch-dependent and Notch-independent populations.Expression of the int-1 and int-2 loci in endogenous mouse mammary tumor virus-induced mammary tumorigenesis in the C3Hf mouse.Fim-1, Fim-2/c-fms, and Fim-3, three common integration sites of Friend murine leukemia virus in myeloblastic leukemias, map to mouse chromosomes 13, 18, and 3, respectivelyThe Notch signaling pathway as a mediator of tumor survivalLessons Learned from Mouse Mammary Tumor Virus in Animal Models.Notch3 inhibits epithelial-mesenchymal transition by activating Kibra-mediated Hippo/YAP signaling in breast cancer epithelial cells.Suppression of mouse mammary tumor proviral DNA and protooncogene expression: association with nutritional regulation of mammary tumor development.Insertional oncogenesis by non-acute retroviruses: implications for gene therapy.Anti-DLL4, a cancer therapeutic with multiple mechanisms of action.Molecular mechanisms of asymmetric divisions in mammary stem cells.Neoplastic transformation by Notch requires nuclear localizationA putative int domain for mouse mammary tumor virus on mouse chromosome 7 is a 5' extension of int-2.
P2860
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P2860
A new common integration region (int-3) for mouse mammary tumor virus on mouse chromosome 17.
description
1987 nî lūn-bûn
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1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
A new common integration regio ...... virus on mouse chromosome 17.
@en
type
label
A new common integration regio ...... virus on mouse chromosome 17.
@en
prefLabel
A new common integration regio ...... virus on mouse chromosome 17.
@en
P2093
P2860
P1433
P1476
A new common integration regio ...... virus on mouse chromosome 17.
@en
P2093
P2860
P304
P407
P577
1987-01-01T00:00:00Z