Biological consequences of neuraminidase deficiency in Newcastle disease virus.
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Contribution of the human parainfluenza virus type 3 HN-receptor interaction to pathogenesis in vivo.Monoclonal antibodies to hemagglutinin-neuraminidase and fusion glycoproteins of Newcastle disease virus: relationship between glycosylation and reactivity.The hemagglutinin-neuraminidase protein of Newcastle disease virus determines tropism and virulence.Fixation of oligosaccharides to a surface may increase the susceptibility to human parainfluenza virus 1, 2, or 3 hemagglutinin-neuraminidase.Fusion mutants of Newcastle disease virus selected with monoclonal antibodies to the hemagglutinin-neuraminidase.
P2860
Biological consequences of neuraminidase deficiency in Newcastle disease virus.
description
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1983年の論文
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name
Biological consequences of neuraminidase deficiency in Newcastle disease virus.
@en
type
label
Biological consequences of neuraminidase deficiency in Newcastle disease virus.
@en
prefLabel
Biological consequences of neuraminidase deficiency in Newcastle disease virus.
@en
P2860
P1433
P1476
Biological consequences of neuraminidase deficiency in Newcastle disease virus.
@en
P2093
L E Hightower
P2860
P304
P407
P577
1983-09-01T00:00:00Z