The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction. - Wikidata - wikimedia - TriplyDB

The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

http://www.wikidata.org/entity/Q36909423

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Recent insights into the molecular mechanisms of the NLRP3 inflammasome activation Minimal Invasive Cystometry and Intra-Abdominal Pressure Assessments in Rodents: A Novel Animal Study.The NACHT, LRR and PYD Domains-Containing Protein 3 (NLRP3) Inflammasome Mediates Inflammation and Voiding Dysfunction in a Lipopolysaccharide-Induced Rat Model of Cystitis.Inflammasomes in the urinary tract: a disease-based review.Immunity to uropathogens: the emerging roles of inflammasomes.New frontiers in molecular and imaging research on evaluation and diagnosis of bladder outlet obstruction in women.Bladder fibrosis during outlet obstruction is triggered through the NLRP3 inflammasome and the production of IL-1β.NLRP3/IL-1β mediates denervation during bladder outlet obstruction in rats.Role of NLRP3 Inflammasome in Eosinophilic and Non-eosinophilic Chronic Rhinosinusitis with Nasal Polyps.The Emerging Role of Inflammasomes as Central Mediators in Inflammatory Bladder Pathology.

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The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

http://www.wikidata.org/entity/Q36909423

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

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The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

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The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

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J.JURO.2015.12.068

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The Journal of Urology

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The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction

@en

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Aliya Dumas

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Andrew T Edmondson

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Case M Wood

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Francis M Hughes

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Hayden M Hill

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J Todd Purves

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James M Oelsen

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Wen-Chi Foo

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P2860

Evolutionary medicine and chronic inflammatory state--known and new concepts in pathophysiology

Inflammasomes are important mediators of cyclophosphamide-induced bladder inflammation.

Mechanism of NLRP3 inflammasome activation

NLRP3 inflammasome: from a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases.

Early sequential changes in bladder function after partial bladder outlet obstruction in awake sprague-dawley rats: focus on the decompensated bladder.

The potential repertoire of the innate immune system in the bladder: expression of pattern recognition receptors in the rat bladder and a rat urothelial cell line (MYP3 cells).

Glyburide inhibits the Cryopyrin/Nalp3 inflammasome.

Cyclophosphamide Induces an Early Wave of Acrolein-Independent Apoptosis in the Urothelium.

Necrotic cells trigger a sterile inflammatory response through the Nlrp3 inflammasome.

Functional consequences of chronic bladder ischemia.

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