Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts
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USP4 inhibits p53 through deubiquitinating and stabilizing ARF-BP1PML, a growth suppressor disrupted in acute promyelocytic leukemiaA group of type I keratin genes on human chromosome 17: characterization and expressionIsolation, sequence, and differential expression of a human K7 gene in simple epithelial cellsRB and c-Myc activate expression of the E-cadherin gene in epithelial cells through interaction with transcription factor AP-2.Transcriptional activation by Myc is under negative control by the transcription factor AP-2SIRT3 is a mitochondria-localized tumor suppressor required for maintenance of mitochondrial integrity and metabolism during stressIsolation, sequence, and expression of a human keratin K5 gene: transcriptional regulation of keratins and insights into pairwise controlExpression of sprouty2 inhibits B-cell proliferation and is epigenetically silenced in mouse and human B-cell lymphomasThe expression of mutant epidermal keratin cDNAs transfected in simple epithelial and squamous cell carcinoma linesHepatitis C virus core from two different genotypes has an oncogenic potential but is not sufficient for transforming primary rat embryo fibroblasts in cooperation with the H-ras oncogene.Hepatitis C virus core protein cooperates with ras and transforms primary rat embryo fibroblasts to tumorigenic phenotypeATP-stimulated c-fos and zif268 mRNA expression is inhibited by chemical hypoxia in a rat brain-derived type 2 astrocyte cell line, RBA-2Deletion of Mnt leads to disrupted cell cycle control and tumorigenesisRecessive mechanisms of malignancy.Antiproliferative properties of the USF family of helix-loop-helix transcription factorsOncogene v-src transforms and establishes embryonic rodent fibroblasts but not diploid human fibroblasts.Genetic analysis of indefinite division in human cells: identification of four complementation groups.Malignant transformation of mouse primary keratinocytes by Harvey sarcoma virus and its modulation by surrounding normal cells.Sustained expression of the human protooncogene MYCN rescues rat embryo cells from senescence.Tumors induced in mice by direct inoculation of plasmid DNA expressing both activated H-ras and c-myc.Sequential expression and cooperative interaction of c-Ha-ras and c-erbB genes in in vivo chemical carcinogenesis.The ras and myc oncogenes cooperate in tumor induction in many tissues when introduced into midgestation mouse embryos by retroviral vectors.Ras-mediated cell cycle arrest is altered by nuclear oncogenes to induce Schwann cell transformation.Adaptation to hypoxia and acidosis in carcinogenesis and tumor progressionAmplification and rearrangement of the Kirsten ras oncogene in virus-transformed BALB/c 3T3 cells during malignant tumor progression.Development of transforming function during transduction of proto-ras into Harvey sarcoma virus.Mechanisms of oncogene cooperation: activation and inactivation of a growth antagonist.A new approach to the molecular basis of neoplastic transformation in the brain.The c-MYC oncoprotein as a treatment target in cancer and other disorders of cell growth.Metal-inducible pathology in the liver, pancreas, and kidney of transgenic mice expressing SV40 early region genes.Characterization of Leukemia-Inducing Genes Using a Proto-Oncogene/Homeobox Gene Retroviral Human cDNA Library in a Mouse In Vivo Model.Cellular aging is a critical determinant of primary cell resistance to v-src transformation.SIRT3 is a mitochondrial tumor suppressor: a scientific tale that connects aberrant cellular ROS, the Warburg effect, and carcinogenesisAnalysis of the tumorigenic potential of common marmoset lymphoblastoid cells expressing a constitutively activated c-myc gene.Can ends justify the means?: telomeres and the mechanisms of replicative senescence and immortalization in mammalian cellsExpression of mutant keratin cDNAs in epithelial cells reveals possible mechanisms for initiation and assembly of intermediate filamentsv-src transformation of rat embryo fibroblasts. Inefficient conversion to anchorage-independent growth involves heterogeneity of primary culturesThe mdm-2 oncogene can overcome wild-type p53 suppression of transformed cell growth.c-myc reverses neu-induced transformed morphology by transcriptional repression.
P2860
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P2860
Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
1986年论文
@zh
1986年论文
@zh-cn
name
Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts
@en
type
label
Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts
@en
prefLabel
Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts
@en
P2093
P2860
P356
P1476
Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts
@en
P2093
J P Morgenstern
L F Parada
R A Weinberg
P2860
P304
P356
10.1128/MCB.6.6.1917
P407
P577
1986-06-01T00:00:00Z