Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia. - Wikidata - wikimedia - TriplyDB

Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia.

Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia.

http://www.wikidata.org/entity/Q36916418

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Treatment of Diabetes and Diabetic Complications With a Ketogenic Diet Glucose stimulates cholesterol 7alpha-hydroxylase gene transcription in human hepatocytes The role of epigenetics in the pathology of diabetic complications Activation of PKC-delta and SHP-1 by hyperglycemia causes vascular cell apoptosis and diabetic retinopathy Epigenetics: a molecular link between environmental factors and type 2 diabetes The Effectiveness of Different Diet Strategies to Reduce Type 2 Diabetes Risk in Youth Histone Acetylation and Its Modifiers in the Pathogenesis of Diabetic Nephropathy Endothelial Progenitor Cells in Diabetic Microvascular Complications: Friends or Foes?Cardiovascular Outcome Studies in Diabetes: How Do We Make Sense of These New Data?Perspectives on systems biology applications in diabetic kidney disease Diabetes and cardiovascular disease: changing the focus from glycemic control to improving long-term survival Nitric oxide, oxidative stress, and p66Shc interplay in diabetic endothelial dysfunction Endothelial dysfunction in diabetes mellitus: possible involvement of endoplasmic reticulum stress?The emerging role of epigenetics in cardiovascular disease Epigenetic Changes in Endothelial Progenitors as a Possible Cellular Basis for Glycemic Memory in Diabetic Vascular Complications Human genetics of diabetic vascular complications Identifying novel transcriptional components controlling energy metabolism Molecular mechanisms of diabetic kidney disease Transcriptional regulation by the Set7 lysine methyltransferase Insulin resistance, hyperglycemia, and atherosclerosis Epigenomics: the science of no-longer-junk DNA. Why study it in chronic kidney disease?Alteration of energy substrates and ROS production in diabetic cardiomyopathy Transcriptional regulation of endothelial dysfunction in atherosclerosis: an epigenetic perspective Chromatin Modifications Associated With Diabetes and Obesity Molecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models.Diabetes propels the risk for cardiovascular disease: sweet monocytes becoming aggressive?Considering maternal dietary modulators for epigenetic regulation and programming of the fetal epigenome p75(NTR)-dependent activation of NF-κB regulates microRNA-503 transcription and pericyte-endothelial crosstalk in diabetes after limb ischaemia Mice lacking the β2 adrenergic receptor have a unique genetic profile before and after focal brain ischaemia Mitochondrial hormesis and diabetic complications Histone variants and their post-translational modifications in primary human fat cells Epigenetic modification of Sod2 in the development of diabetic retinopathy and in the metabolic memory: role of histone methylation Epigenetic histone methylation modulates fibrotic gene expression Mitochondria DNA replication and DNA methylation in the metabolic memory associated with continued progression of diabetic retinopathy Oxidative stress and diabetic complications Defining a research agenda to address the converging epidemics of tuberculosis and diabetes. Part 2: Underlying biological mechanisms.Differential Response to High Glucose in Skin Fibroblasts of Monozygotic Twins Discordant for Type 1 Diabetes Gene expression differences in skin fibroblasts in identical twins discordant for type 1 diabetes.Child health, developmental plasticity, and epigenetic programming Diabetes impairs hematopoietic stem cell mobilization by altering niche function.

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P2860

Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia.

http://www.wikidata.org/entity/Q36916418

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Transient high glucose causes ...... ring subsequent normoglycemia.

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type

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Transient high glucose causes ...... ring subsequent normoglycemia.

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Transient high glucose causes ...... ring subsequent normoglycemia.

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Transient high glucose causes ...... ring subsequent normoglycemia.

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Alessandro Pocai

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Dachun Yao

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Daniella Brasacchio

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Mark E Cooper

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Michael Brownlee

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Robert G Roeder

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P2860

Intensive Diabetes Treatment and Cardiovascular Disease in Patients with Type 1 Diabetes

Brahma links the SWI/SNF chromatin-remodeling complex with MeCP2-dependent transcriptional silencing

Overexpression of glyoxalase-I in bovine endothelial cells inhibits intracellular advanced glycation endproduct formation and prevents hyperglycemia-induced increases in macromolecular endocytosis

High-resolution profiling of histone methylations in the human genome

Chromatin modifications and their function

Biochemistry and molecular cell biology of diabetic complications

Diabetes-associated sustained activation of the transcription factor nuclear factor-kappaB

Genomic DNA methylation: the mark and its mediators

Genetic and epigenetic factors are associated with expression of respiratory chain component NDUFB6 in human skeletal muscle

Regulation of MLL1 H3K4 methyltransferase activity by its core components

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