MUC5B Is the predominant mucin glycoprotein in chronic otitis media fluid.
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Otitis media in a new mouse model for CHARGE syndrome with a deletion in the Chd7 geneMuc5b is required for airway defenceEvaluation of 15 functional candidate genes for association with chronic otitis media with effusion and/or recurrent otitis media (COME/ROM).Panel 4: Recent advances in otitis media in molecular biology, biochemistry, genetics, and animal models.In vivo imaging of the Muc5b gel-forming mucinOtitis media in sperm-associated antigen 6 (Spag6)-deficient mice.Panel 3: Recent advances in anatomy, pathology, and cell biology in relation to otitis media pathogenesis.SuperSILAC Quantitative Proteome Profiling of Murine Middle Ear Epithelial Cell Remodeling with NTHi.Proteomic Characterization of Middle Ear Fluid Confirms Neutrophil Extracellular Traps as a Predominant Innate Immune Response in Chronic Otitis MediaQuantitative proteomics reveals an altered cystic fibrosis in vitro bronchial epithelial secretomeSalivary mucins in host defense and disease preventionAdvances in the proteomic investigation of the cell secretome.A proteomic characterization of NTHi lysatesMucin Agarose Gel Electrophoresis: Western Blotting for High-molecular-weight Glycoproteins.NTHi induction of Cxcl2 and middle ear mucosal metaplasia in mice.The human secretome atlas initiative: implications in health and disease conditions.An in vitro model of murine middle ear epithelium.Directional secretomes reflect polarity-specific functions in an in vitro model of human bronchial epithelium.Mucin production and mucous cell metaplasia in otitis media.Current knowledge of the genetics of otitis media.Panel 1: Epidemiology, natural history, and risk factors.Proteomic analysis of pediatric sinonasal secretions shows increased MUC5B mucin in CRS.Association of Gel-Forming Mucins and Aquaporin Gene Expression With Hearing Loss, Effusion Viscosity, and Inflammation in Otitis Media With Effusion.Impact of Staphylococcus epidermidis lysates on middle ear epithelial proinflammatory and mucogenic response.Younger patients with COME are more likely to have mucoid middle ear fluid containing mucin MUC5B.Differential response of gel-forming mucins to pathogenic middle ear bacteria.Purification and characterization of microRNAs within middle ear fluid exosomes: implication in otitis media pathophysiology.Multi-scale finite element modeling of Eustachian tube function: influence of mucosal adhesion.Nontypeable Haemophilus influenzae lysates increase heterogeneous nuclear ribonucleoprotein secretion and exosome release in human middle-ear epithelial cells.Loss of the homeostatic protein BPIFA1, leads to exacerbation of otitis media severity in the Junbo mouse model.Multi-scale modeling of an upper respiratory airway: Effect of mucosal adhesion on Eustachian tube function in young children.
P2860
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P2860
MUC5B Is the predominant mucin glycoprotein in chronic otitis media fluid.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
MUC5B Is the predominant mucin glycoprotein in chronic otitis media fluid.
@en
type
label
MUC5B Is the predominant mucin glycoprotein in chronic otitis media fluid.
@en
prefLabel
MUC5B Is the predominant mucin glycoprotein in chronic otitis media fluid.
@en
P2093
P2860
P1433
P1476
MUC5B Is the predominant mucin glycoprotein in chronic otitis media fluid
@en
P2093
Alan M Watson
Diego Preciado
Mary C Rose
Michael Rahimi
Samita Goyal
Yetrib Hathout
P2860
P2888
P304
P356
10.1203/PDR.0B013E3181EB2ECC
P407
P577
2010-09-01T00:00:00Z
P6179
1005790642