Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice - Wikidata - wikimedia - TriplyDB

Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

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Hypersensitivity of Vagal Pulmonary Afferents Induced by Tumor Necrosis Factor Alpha in Mice.Hemorrhagic hypotension-induced hypersensitivity of vagal pulmonary C-fibers to chemical stimulation and lung inflation in anesthetized rats.Hypersensitivity of vagal pulmonary C-fibers induced by increasing airway temperature in ovalbumin-sensitized rats Investigation into the Role of PI3K and JAK3 Kinase Inhibitors in Murine Models of Asthma.Sustained sensitizing effects of tumor necrosis factor alpha on sensory nerves in lung and airways.Phrenic motor outputs in response to bronchopulmonary C-fibre activation following chronic cervical spinal cord injury.Immediate and delayed potentiating effects of tumor necrosis factor-α on TRPV1 sensitivity of rat vagal pulmonary sensory neurons.Cough and expiration reflexes elicited by inhaled irritant gases are intensified in ovalbumin-sensitized mice.Recovery of the pulmonary chemoreflex and functional role of bronchopulmonary C-fibers following chronic cervical spinal cord injury.

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Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

http://www.wikidata.org/entity/Q36924182

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2013 nî lūn-bûn

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name

Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

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Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

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Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

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JAPPLPHYSIOL.01301.2012

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Journal of Applied Physiology

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Pulmonary chemoreflex responses are potentiated by tumor necrosis factor-alpha in mice

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Lu-Yuan Lee

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Marcus J Geer

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Richard Kryscio

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Ruei-Lung Lin

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Yu-Jung Lin

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P2860

Pungent products from garlic activate the sensory ion channel TRPA1

Pathophysiology of the cysteinyl leukotrienes and effects of leukotriene receptor antagonists in asthma

Interleukin-4, -5, and -6 and tumor necrosis factor-alpha in normal and asthmatic airways: evidence for the human mast cell as a source of these cytokines

Bioactive lipoxygenase metabolites stimulation of NADPH oxidases and reactive oxygen species

Direct activation of capsaicin receptors by products of lipoxygenases: endogenous capsaicin-like substances

Afferent properties and reflex functions of bronchopulmonary C-fibers.

Cytokines in asthma.

Bradykinin-12-lipoxygenase-VR1 signaling pathway for inflammatory hyperalgesia.

Tumour necrosis factor-alpha: the role of this multifunctional cytokine in asthma.

Pentraxin 3, a non-redundant soluble pattern recognition receptor involved in innate immunity.

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1536-1543

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10.1152/JAPPLPHYSIOL.01301.2012

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23539315

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3680829

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