Activation of TLR3 in keratinocytes increases expression of genes involved in formation of the epidermis, lipid accumulation, and epidermal organelles.
about
Poly I:C induces collective migration of HaCaT keratinocytes via IL-8.Toll-like receptor 3 activation is required for normal skin barrier repair following UV damage.PHF11 expression and cellular distribution is regulated by the Toll-Like Receptor 3 Ligand Polyinosinic:Polycytidylic Acid in HaCaT keratinocytes.Non-coding Double-stranded RNA and Antimicrobial Peptide LL-37 Induce Growth Factor Expression from Keratinocytes and Endothelial Cells.Antimicrobial Peptide LL37 and MAVS Signaling Drive Interferon-β Production by Epidermal Keratinocytes during Skin Injury.The Parathyroid Hormone Second Receptor PTH2R and its Ligand Tuberoinfundibular Peptide of 39 Residues TIP39 Regulate Intracellular Calcium and Influence Keratinocyte Differentiation.Non-Coding RNAs: New Players in Skin Wound HealingThe non-coding skin: exploring the roles of long non-coding RNAs in epidermal homeostasis and disease.Molecular biology of atopic dermatitis.UVB radiation illuminates the role of TLR3 in the epidermis.The release of pro-inflammatory cytokines is mediated via mitogen-activated protein kinases rather than by the inflammasome signalling pathway in keratinocytes.TLR3 drives IRF6-dependent IL-23p19 expression and p19/EBI3 heterodimer formation in keratinocytes.Ixodes tick saliva suppresses the keratinocyte cytokine response to TLR2/TLR3 ligands during early exposure to Lyme borreliosis.Toll pathway is required for wound-induced expression of barrier repair genes in the Drosophila epidermisThe parathyroid hormone family member TIP39 interacts with sarco/endoplasmic reticulum Ca2+ - ATPase activity by influencing calcium homoeostasis.Activation of Parathyroid Hormone 2 Receptor Induces Decorin Expression and Promotes Wound Repair.Crystallinity of Double-Stranded RNA-Antimicrobial Peptide Complexes Modulates Toll-Like Receptor 3-Mediated Inflammation.Naturally occurring ω-Hydroxyacids.Polyinosinic-polycytidylic acid (poly(I:C)) attenuates imiquimod-induced skin inflammation in mice by increasing cutaneous PD-L1 expression.After Skin Wounding, Noncoding dsRNA Coordinates Prostaglandins and Wnts to Promote Regeneration.
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P2860
Activation of TLR3 in keratinocytes increases expression of genes involved in formation of the epidermis, lipid accumulation, and epidermal organelles.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Activation of TLR3 in keratino ...... ion, and epidermal organelles.
@en
type
label
Activation of TLR3 in keratino ...... ion, and epidermal organelles.
@en
prefLabel
Activation of TLR3 in keratino ...... ion, and epidermal organelles.
@en
P2093
P2860
P356
P1476
Activation of TLR3 in keratino ...... ion, and epidermal organelles.
@en
P2093
Andrew W Borkowski
Kyungho Park
Richard L Gallo
Yoshikazu Uchida
P2860
P2888
P304
P356
10.1038/JID.2013.39
P407
P577
2013-02-28T00:00:00Z
P5875
P6179
1050178505