TSGΔ154-1054 splice variant increases TSG101 oncogenicity by inhibiting its E3-ligase-mediated proteasomal degradation.

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TSGΔ154-1054 splice variant increases TSG101 oncogenicity by inhibiting its E3-ligase-mediated proteasomal degradation.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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TSGΔ154-1054 splice variant in ...... iated proteasomal degradation.

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TSGΔ154-1054 splice variant in ...... iated proteasomal degradation.

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prefLabel

TSGΔ154-1054 splice variant in ...... iated proteasomal degradation.

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P1476

TSGΔ154-1054 splice variant in ...... diated proteasomal degradation

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P2093

Huey-Huey Chua

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Pei-Lun Weng

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Te-Huei Yeh

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P2860

Tal, a Tsg101-specific E3 ubiquitin ligase, regulates receptor endocytosis and retrovirus budding

Regulation of human p53 activity and cell localization by alternative splicing

Regulation of Tsg101 expression by the steadiness box: a role of Tsg101-associated ligase

A TSG101/MDM2 regulatory loop modulates MDM2 degradation and MDM2/p53 feedback control

Advanced glycation end-products: a biological consequence of lifestyle contributing to cancer disparity

Tumor susceptibility gene 101 protein represses androgen receptor transactivation and interacts with p300

RETRACTED: The TSG101 tumor susceptibility gene is located in chromosome 11 band p15 and is mutated in human breast cancer

Parallels between cytokinesis and retroviral budding: a role for the ESCRT machinery

Targeted deletion of the Tsg101 gene results in cell cycle arrest at G1/S and p53-independent cell death

p53 accumulation, defective cell proliferation, and early embryonic lethality in mice lacking tsg101.

P304

8240-8252

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P31

scholarly article

P356

10.18632/ONCOTARGET.6973

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P407

English

P433

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P478

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P50

Chiun-Sheng Huang

P577

2016-02-01T00:00:00Z

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P698

26811492

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P932

4884989

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