Lymphoid precursors are directed to produce dendritic cells as a result of TLR9 ligation during herpes infection - Wikidata - wikimedia - TriplyDB

Lymphoid precursors are directed to produce dendritic cells as a result of TLR9 ligation during herpes infection

Lymphoid precursors are directed to produce dendritic cells as a result of TLR9 ligation during herpes infection

http://www.wikidata.org/entity/Q36948602

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TLRs control hematopoiesis during infection Infection-induced changes in hematopoiesis Stress and Non-Stress Roles of Inflammatory Signals during HSC Emergence and Maintenance Impact of Viral Infections on Hematopoiesis: From Beneficial to Detrimental Effects on Bone Marrow Output Candida albicans induces selective development of macrophages and monocyte derived dendritic cells by a TLR2 dependent signalling The dendritic cell lineage: ontogeny and function of dendritic cells and their subsets in the steady state and the inflamed setting Transient activation of hematopoietic stem and progenitor cells by IFNγ during acute bacterial infection.The density of CD10 corresponds to commitment and progression in the human B lymphoid lineage.Herpes simplex type I (HSV-1) infection of the nervous system: is an immune response a good thing?Functional diversity of stem and progenitor cells with B-lymphopoietic potential Chronic exposure to a TLR ligand injures hematopoietic stem cells.Separation of plasmacytoid dendritic cells from B-cell-biased lymphoid progenitor (BLP) and Pre-pro B cells using PDCA-1.Lack of the ubiquitin-editing enzyme A20 results in loss of hematopoietic stem cell quiescence Infection-induced myelopoiesis during intracellular bacterial infection is critically dependent upon IFN-γ signaling.Treatment of chronic myelogenous leukemia by blocking cytokine alterations found in normal stem and progenitor cells Bone Marrow Cells in Acute Lymphoblastic Leukemia Create a Proinflammatory Microenvironment Influencing Normal Hematopoietic Differentiation Fates Lineage switching in acute leukemias: a consequence of stem cell plasticity?Early sensing of Yersinia pestis airway infection by bone marrow cells.TLR9 bone marrow chimeric mice define a role for cerebral TNF in neuroprotection induced by CpG preconditioning.Encephalitozoon intestinalis Inhibits Dendritic Cell Differentiation through an IL-6-Dependent Mechanism The development of adult innate lymphoid cells.Obesity-induced DNA released from adipocytes stimulates chronic adipose tissue inflammation and insulin resistance C/EBPα is required for development of dendritic cell progenitors.Innate pro-B-cell progenitors protect against type 1 diabetes by regulating autoimmune effector T cells Toll-like receptor 9 and interferon-γ receptor signaling suppress the B-cell fate of uncommitted progenitors in mice Treatment of ongoing autoimmune encephalomyelitis with activated B-cell progenitors maturing into regulatory B cells.Canonical Wnt pathway signaling suppresses VCAM-1 expression by marrow stromal and hematopoietic cells.Modeling the Pro-inflammatory Tumor Microenvironment in Acute Lymphoblastic Leukemia Predicts a Breakdown of Hematopoietic-Mesenchymal Communication Networks TLR stimulation of bone marrow lymphoid precursors from childhood acute leukemia modifies their differentiation potentials.Lymphoid progenitor cells from childhood acute lymphoblastic leukemia are functionally deficient and express high levels of the transcriptional repressor Gfi-1 Selective expansion of the monocytic lineage directed by bacterial infection Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease.Early Differentiation of Human CD11c+NK Cells with γδ T Cell Activation Properties Is Promoted by Dialyzable Leukocyte Extracts.Systemic Expression of Notch Ligand Delta-Like 4 during Mycobacterial Infection Alters the T Cell Immune Response.Asynchronous RAG-1 expression during B lymphopoiesis.The Role of the Bone Marrow Stromal Compartment in the Hematopoietic Response to Microbial Infections.TLR signaling in B-cell development and activation.T-cell lineage determination.Inflammatory signals regulate hematopoietic stem cells Complexity of dendritic cell subsets and their function in the host immune system.

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Lymphoid precursors are directed to produce dendritic cells as a result of TLR9 ligation during herpes infection

http://www.wikidata.org/entity/Q36948602

description

2008 nî lūn-bûn

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Lymphoid precursors are direct ...... gation during herpes infection

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Lymphoid precursors are direct ...... gation during herpes infection

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Daniel J Carr

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Karla P Garrett

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Lisa A Borghesi

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Paul W Kincade

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Robert S Welner

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Rosana Pelayo

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Todd R Wuest

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Yoshinori Nagai

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P2860

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

Lymphoid neo-organogenesis: lymphotoxin's role in inflammation and development.

Steady-state and inflammatory dendritic-cell development.

Lethal encephalitis in myeloid differentiation factor 88-deficient mice infected with herpes simplex virus 1.

Toll-like receptors on hematopoietic progenitor cells stimulate innate immune system replenishment

Dendritic cells: controllers of the immune system and a new promise for immunotherapy.

Interferon-producing killer dendritic cells (IKDCs) arise via a unique differentiation pathway from primitive c-kitHiCD62L+ lymphoid progenitors.

Inflammation controls B lymphopoiesis by regulating chemokine CXCL12 expression.

Herpes simplex virus 1 interaction with Toll-like receptor 2 contributes to lethal encephalitis.

Innate recognition network driving herpes simplex virus-induced corneal immunopathology: role of the toll pathway in early inflammatory events in stromal keratitis

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9

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Michael A Farrar

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5303158

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18552210

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Toll-like receptor

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2572801

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