Region between the canine distemper virus M and F genes modulates virulence by controlling fusion protein expression.
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Constraints on the Genetic and Antigenic Variability of Measles VirusIllumination of parainfluenza virus infection and transmission in living animals reveals a tissue-specific dichotomyCommon position of indels that cause deviations from canonical genome organization in different measles virus strains.Complete genome sequence of avian paramyxovirus (APMV) serotype 5 completes the analysis of nine APMV serotypes and reveals the longest APMV genome.Residues in the heptad repeat a region of the fusion protein modulate the virulence of Sendai virus in miceElements in the canine distemper virus M 3' UTR contribute to control of replication efficiency and virulenceThe deletion of an extra six nucleotides in the 5' -untranslated region of the nucleoprotein gene of Newcastle disease virus NA-1 decreases virulence.Key genomic changes necessary for an in vivo lethal mouse marburgvirus variant selection process.Wild-type measles viruses with non-standard genome lengths.Complete genome sequence of avian paramyxovirus type 7 (strain Tennessee) and comparison with other paramyxoviruses.Canine distemper virus in the Serengeti ecosystem: molecular adaptation to different carnivore species.Influence of antigen insertion site and vector dose on immunogenicity and protective capacity in Sendai virus-based human parainfluenza virus type 3 vaccines.The structurally disordered paramyxovirus nucleocapsid protein tail domain is a regulator of the mRNA transcription gradient.Morbillivirus control of the interferon response: relevance of STAT2 and mda5 but not STAT1 for canine distemper virus virulence in ferrets.Virulence of Newcastle disease virus: what is known so far?Sequencing of emerging canine distemper virus strain reveals new distinct genetic lineage in the United States associated with disease in wildlife and domestic canine populations.Canine distemper virus epithelial cell infection is required for clinical disease but not for immunosuppression.Canine distemper virus matrix protein influences particle infectivity, particle composition, and envelope distribution in polarized epithelial cells and modulates virulence.Eukaryotic elongation factor 1-beta interacts with the 5' untranslated region of the M gene of Nipah virus to promote mRNA translation.Genome characterization and phylogenetic analysis of a lineage IV peste des petits ruminants virus in southern China.Membrane fusion-mediated autophagy induction enhances morbillivirus cell-to-cell spread.Canine distemper viruses expressing a hemagglutinin without N-glycans lose virulence but retain immunosuppression.Translational enhancing activity in 5' UTR of peste des petits ruminants virus fusion gene.The Unstructured Paramyxovirus Nucleocapsid Protein Tail Domain Modulates Viral Pathogenesis through Regulation of Transcriptase Activity.
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P2860
Region between the canine distemper virus M and F genes modulates virulence by controlling fusion protein expression.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Region between the canine dist ...... ing fusion protein expression.
@en
type
label
Region between the canine dist ...... ing fusion protein expression.
@en
prefLabel
Region between the canine dist ...... ing fusion protein expression.
@en
P2860
P356
P1433
P1476
Region between the canine dist ...... ing fusion protein expression.
@en
P2093
Danielle E Anderson
Veronika von Messling
P2860
P304
10510-10518
P356
10.1128/JVI.01419-08
P407
P577
2008-08-27T00:00:00Z