Changes in action potentials and intracellular ionic homeostasis in a ventricular cell model related to a persistent sodium current in SCN5A mutations underlying LQT3.
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Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutationUnusual case of severe arrhythmia developed after acute intoxication with tosylchloramide.Human Na(v)1.8: enhanced persistent and ramp currents contribute to distinct firing properties of human DRG neurons.The arrhythmogenic consequences of increasing late INa in the cardiomyocyte.Contribution of the late sodium current to intracellular sodium and calcium overload in rabbit ventricular myocytes treated by anemone toxin.
P2860
Changes in action potentials and intracellular ionic homeostasis in a ventricular cell model related to a persistent sodium current in SCN5A mutations underlying LQT3.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Changes in action potentials a ...... N5A mutations underlying LQT3.
@en
type
label
Changes in action potentials a ...... N5A mutations underlying LQT3.
@en
prefLabel
Changes in action potentials a ...... N5A mutations underlying LQT3.
@en
P2093
P1476
Changes in action potentials a ...... CN5A mutations underlying LQT3
@en
P2093
P304
P356
10.1016/J.PBIOMOLBIO.2007.07.023
P577
2007-08-11T00:00:00Z