Alternatively activated macrophages determine repair of the infarcted adult murine heart. - Wikidata - wikimedia - TriplyDB

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

http://www.wikidata.org/entity/Q36951782

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Getting to the heart of intracellular glucocorticoid regeneration: 11β-HSD1 in the myocardium Role of Macrophages in the Repair Process during the Tissue Migrating and Resident Helminth Infections Cell Size Critically Determines Initial Retention of Bone Marrow Mononuclear Cells in the Heart after Intracoronary Injection: Evidence from a Rat Model Cardiosphere-Derived Cells Facilitate Heart Repair by Modulating M1/M2 Macrophage Polarization and Neutrophil Recruitment.Harnessing the early post-injury inflammatory responses for cardiac regeneration.Macrophage activation and polarization in post-infarction cardiac remodeling.Diabetic Cardiomyopathy: An Immunometabolic Perspective.Myeloid receptor CD36 is required for early phagocytosis of myocardial infarcts and induction of Nr4a1-dependent mechanisms of cardiac repair.IL-4 as a Repurposed Biological Drug for Myocardial Infarction through Augmentation of Reparative Cardiac Macrophages: Proof-of-Concept Data in Mice.Heart regeneration and repair after myocardial infarction: translational opportunities for novel therapeutics.Topiramate modulates post-infarction inflammation primarily by targeting monocytes or macrophages.Prolyl-hydroxylase inhibition induces SDF-1 associated with increased CXCR4+/CD11b+ subpopulations and cardiac repair Recruited Monocytes and Type 2 Immunity Promote Lung Regeneration following Pneumonectomy.Macrophages and skeletal health.Macrophage-based therapeutic strategies in regenerative medicine.The Role of Cardiac Tissue Macrophages in Homeostasis and Disease.Ectonucleotidase CD39-driven control of postinfarction myocardial repair and rupture.Loss of Macrophage Wnt Secretion Improves Remodeling and Function After Myocardial Infarction in Mice.Long-Term Administration of Eicosapentaenoic Acid Improves Post-Myocardial Infarction Cardiac Remodeling in Mice by Regulating Macrophage Polarization.Periodontal-induced chronic inflammation triggers macrophage secretion of Ccl12 to inhibit fibroblast-mediated cardiac wound healing.Efferocytosis and Outside-In Signaling by Cardiac Phagocytes. Links to Repair, Cellular Programming, and Intercellular Crosstalk in Heart.Apoptosis inhibitor of macrophage depletion decreased M1 macrophage accumulation and the incidence of cardiac rupture after myocardial infarction in mice.Cardiac macrophage biology in the steady-state heart, the aging heart, and following myocardial infarction.Replenishing the damaged heart with oxygen by nature-inspired photosynthesis.Anti-inflammatory therapies in myocardial infarction: failures, hopes, and challenges.Ac-SDKP decreases mortality and cardiac rupture after acute myocardial infarction.Cardiac macrophages promote diastolic dysfunction.Macrophage Transitions in Heart Valve Development and Myxomatous Valve Disease.Cytosolic DNA Sensing Promotes Macrophage Transformation and Governs Myocardial Ischemic Injury.Adenosine Production by Biomaterial-Supported Mesenchymal Stromal Cells Reduces the Innate Inflammatory Response in Myocardial Ischemia/Reperfusion Injury.Mechanisms of Fibroblast Activation in the Remodeling Myocardium.iRhom2-mediated proinflammatory signalling regulates heart repair following myocardial infarction.Immune mechanisms in heart failure.Temporal cascade of inflammatory cytokines and cell-type populations in monocyte chemotactic protein-1 (MCP-1)-mediated aneurysm healing.Morphological and functional analyses of skeletal muscles from an immunodeficient animal model of limb girdle muscular dystrophy type 2E.HDAC inhibition helps post-MI healing by modulating macrophage polarization.The puzzling mechanism of compensatory lung growth.Cardiomyocytes and Macrophages Discourse on the Method to Govern Cardiac Repair The Macrophage in Cardiac Homeostasis and Disease: JACC Macrophage in CVD Series (Part 4)Parallels between vertebrate cardiac and cutaneous wound healing and regeneration

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P2860

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

http://www.wikidata.org/entity/Q36951782

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

@yue

2016年論文

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2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

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2016年论文

@zh

2016年论文

@zh-cn

name

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

@en

type

label

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

@en

prefLabel

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

@en

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Journal of Clinical Investigation

P1476

Alternatively activated macrophages determine repair of the infarcted adult murine heart.

@en

P2093

Atsushi Yamaguchi

http://www.w3.org/2001/XMLSchema#string

Hidekazu Ishida

http://www.w3.org/2001/XMLSchema#string

Hideo Adachi

http://www.w3.org/2001/XMLSchema#string

Ken Suzuki

http://www.w3.org/2001/XMLSchema#string

Kenta Yashiro

http://www.w3.org/2001/XMLSchema#string

Manabu Shiraishi

http://www.w3.org/2001/XMLSchema#string

Rie Saba

http://www.w3.org/2001/XMLSchema#string

Yasunori Shintani

http://www.w3.org/2001/XMLSchema#string

Yusuke Shintani

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P2860

VEGF gene therapy: therapeutic angiogenesis in the clinic and beyond

Critical role of Trib1 in differentiation of tissue-resident M2-like macrophages

Macrophage plasticity and polarization: in vivo veritas

Alternative activation of macrophages: mechanism and functions

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Transient regenerative potential of the neonatal mouse heart

Comitogenic effect of catecholamines on rat cardiac fibroblasts in culture.

Osteopontin: role in extracellular matrix deposition and myocardial remodeling post-MI.

Differential contribution of monocytes to heart macrophages in steady-state and after myocardial infarction.

Lipocalin 2 is a regulator of macrophage polarization and NF-κB/STAT3 pathway activation

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2151-2166

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10.1172/JCI85782

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6

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126

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27140396

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4887176

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