Aggresomes and pericentriolar sites of virus assembly: cellular defense or viral design?
about
Virion assembly factories in the nucleus of polyomavirus-infected cellsA physical interaction network of dengue virus and human proteinsMicrotubules in viral replication and transportCoupling viruses to dynein and kinesin-1Autophagy in hepatitis C virus-host interactions: potential roles and therapeutic targets for liver-associated diseasesRecruitment of the oncoprotein v-ErbA to aggresomes.An intrinsically disordered region of the acetyltransferase p300 with similarity to prion-like domains plays a role in aggregation3D reconstruction of VZV infected cell nuclei and PML nuclear cages by serial section array scanning electron microscopy and electron tomographySeeking Membranes: Positive-Strand RNA Virus Replication ComplexesUbiquitin in Influenza Virus Entry and Innate ImmunityThe herpesvirus VP1/2 protein is an effector of dynein-mediated capsid transport and neuroinvasion.A Rab11- and microtubule-dependent mechanism for cytoplasmic transport of influenza A virus viral RNACyclin-dependent kinase-like function is shared by the beta- and gamma- subset of the conserved herpesvirus protein kinasesA versatile molecular tagging method for targeting proteins to avian reovirus muNS inclusions. Use in protein immobilization and purificationMurine cytomegalovirus capsid assembly is dependent on US22 family gene M140 in infected macrophagesIntracellular transport of the measles virus ribonucleoprotein complex is mediated by Rab11A-positive recycling endosomes and drives virus release from the apical membrane of polarized epithelial cells.Multilamellar structures and filament bundles are found on the cell surface during bunyavirus egress.Enterovirus 71 VP1 activates calmodulin-dependent protein kinase II and results in the rearrangement of vimentin in human astrocyte cells.Entrapment of viral capsids in nuclear PML cages is an intrinsic antiviral host defense against varicella-zoster virus.AAV exploits subcellular stress associated with inflammation, endoplasmic reticulum expansion, and misfolded proteins in models of cystic fibrosis.Functional characterization of Negri bodies (NBs) in rabies virus-infected cells: Evidence that NBs are sites of viral transcription and replication.Virus-induced aggregates in infected cells.Congenital cataract causing mutants of αA-crystallin/sHSP form aggregates and aggresomes degraded through ubiquitin-proteasome pathway.Secretion of the human Toll-like receptor 3 ectodomain is affected by single nucleotide polymorphisms and regulated by Unc93b1.Activation of the unfolded protein response and autophagy after hepatitis C virus infection suppresses innate antiviral immunity in vitro.Aquareovirus NS80 recruits viral proteins to its inclusions, and its C-terminal domain is the primary driving force for viral inclusion formation.The herpes simplex virus immediate-early ubiquitin ligase ICP0 induces degradation of the ICP0 repressor protein E2FBP1.Schizophrenia susceptibility genes directly implicated in the life cycles of pathogens: cytomegalovirus, influenza, herpes simplex, rubella, and Toxoplasma gondii.Foot-and-mouth disease virus, but not bovine enterovirus, targets the host cell cytoskeleton via the nonstructural protein 3Cpro.Recruitment of the host plant heat shock protein 70 by Tomato yellow leaf curl virus coat protein is required for virus infectionSalmonella - at home in the host cell.Bidirectional lipid droplet velocities are controlled by differential binding strengths of HCV core DII protein.The adenovirus E4 11 k protein binds and relocalizes the cytoplasmic P-body component Ddx6 to aggresomes.Aquareovirus NS80 Initiates Efficient Viral Replication by Retaining Core Proteins within Replication-Associated Viral Inclusion Bodies.Mouse norovirus 1 utilizes the cytoskeleton network to establish localization of the replication complex proximal to the microtubule organizing centerSalmonella effectors: important players modulating host cell function during infection.Cellular chaperones and folding enzymes are vital contributors to membrane bound replication and movement complexes during plant RNA virus infection.Formation of telomeric repeat-containing RNA (TERRA) foci in highly proliferating mouse cerebellar neuronal progenitors and medulloblastoma.Mutations in the amino terminus of foamy virus Gag disrupt morphology and infectivity but do not target assembly.Disruption of Microtubules Post-Virus Entry Enhances Adeno-Associated Virus Vector Transduction.
P2860
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P2860
Aggresomes and pericentriolar sites of virus assembly: cellular defense or viral design?
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Aggresomes and pericentriolar sites of virus assembly: cellular defense or viral design?
@en
type
label
Aggresomes and pericentriolar sites of virus assembly: cellular defense or viral design?
@en
prefLabel
Aggresomes and pericentriolar sites of virus assembly: cellular defense or viral design?
@en
P1476
Aggresomes and pericentriolar sites of virus assembly: cellular defense or viral design?
@en
P2093
Thomas Wileman
P304
P356
10.1146/ANNUREV.MICRO.57.030502.090836
P577
2007-01-01T00:00:00Z