Spermatid head retention as a marker of 2,5-hexanedione-induced testicular toxicity in the rat - Wikidata - wikimedia - TriplyDB

Spermatid head retention as a marker of 2,5-hexanedione-induced testicular toxicity in the rat

Spermatid head retention as a marker of 2,5-hexanedione-induced testicular toxicity in the rat

http://www.wikidata.org/entity/Q36952456

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Male germ cell apoptosis: regulation and biology.Suppression of radiation-induced testicular germ cell apoptosis by 2,5-hexanedione pretreatment. I. Histopathological analysis reveals stage dependence of attenuated apoptosis Sperm mRNA transcripts are indicators of sub-chronic low dose testicular injury in the Fischer 344 rat Impairment of hepatic and renal functions by 2,5-hexanedione is accompanied by oxidative stress in rats.Proliferative and nonproliferative lesions of the rat and mouse male reproductive system.Testicular histopathology associated with disruption of the Sertoli cell cytoskeleton.Is toxicant-induced Sertoli cell injury in vitro a useful model to study molecular mechanisms in spermatogenesis?Pre- and postnatal exposure to ambient levels of urban particulate matter (PM(2.5)) affects mice spermatogenesis.From the Cover: Sperm Molecular Biomarkers Are Sensitive Indicators of Testicular Injury following Subchronic Model Toxicant Exposure.Redox status of the testes and sperm of rats following exposure to 2,5-hexanedione.

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Spermatid head retention as a marker of 2,5-hexanedione-induced testicular toxicity in the rat

http://www.wikidata.org/entity/Q36952456

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2008 nî lūn-bûn

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Toxicologic Pathology

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Bronwyn H Bryant

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Hideki Yamasaki

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Kim Boekelheide

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Moses A Sandrof

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P2860

2,5-Hexanedione alters microtubule assembly. I. Testicular atrophy, not nervous system toxicity, correlates with enhanced tubulin polymerization.

2,5-Hexanedione alters microtubule assembly II. Enhanced polymerization of crosslinked tubulin

Rat testis during 2,5-hexanedione intoxication and recovery I. Dose response and the reversibility of germ cell loss

Rat testis during 2,5-hexanedione intoxication and recovery II. Dynamics of pyrrole reactivity, tubulin content, and microtubule assembly

Degeneration of germ cells in normal, hypophysectomized and hormone treated hypophysectomized rats.

Covalent crosslinking of neurofilaments in the pathogenesis of n-hexane neuropathy.

Development of testicular lesions in F344 rats after treatment with boric acid.

Adverse male reproductive effects following subchronic exposure of rats to sodium dichloroacetate.

2,5-Hexanedione exposure alters microtubule motor distribution in adult rat testis.

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