Moderate perinatal arsenic exposure alters neuroendocrine markers associated with depression and increases depressive-like behaviors in adult mouse offspring.
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Arsenic Exposure Induces Unscheduled Mitotic S Phase Entry Coupled with Cell Death in Mouse Cortical AstrocytesIn utero and early life arsenic exposure in relation to long-term health and diseaseSubchronic Arsenic Exposure Induces Anxiety-Like Behaviors in Normal Mice and Enhances Depression-Like Behaviors in the Chemically Induced Mouse Model of DepressionEffects of prenatal exposure to sodium arsenite on motor and food-motivated behaviors from birth to adulthood in C57BL6/J mice.Fluoxetine treatment ameliorates depression induced by perinatal arsenic exposure via a neurogenic mechanismAdult hippocampal neurogenesis and mRNA expression are altered by perinatal arsenic exposure in mice and restored by brief exposure to enrichment.Genome-wide alteration of histone H3K9 acetylation pattern in mouse offspring prenatally exposed to arsenicReduced expression of MAPK/ERK genes in perinatal arsenic-exposed offspring induced by glucocorticoid receptor deficits.Developmental exposure to 50 parts-per-billion arsenic influences histone modifications and associated epigenetic machinery in a region- and sex-specific manner in the adult mouse brainPerinatal exposure to 50 ppb sodium arsenate induces hypothalamic-pituitary-adrenal axis dysregulation in male C57BL/6 micePrenatal arsenic exposure alters the programming of the glucocorticoid signaling system during embryonic development.Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain.Gene-environment interactions affect long-term depression (LTD) through changes in dopamine receptor affinity in Snap25 deficient mice.A systematic review of arsenic exposure and its social and mental health effects with special reference to BangladeshLearning deficits in C57BL/6J mice following perinatal arsenic exposure: consequence of lower corticosterone receptor levels?Cortical Astrocytes Acutely Exposed to the Monomethylarsonous Acid (MMAIII) Show Increased Pro-inflammatory Cytokines Gene Expression that is Consistent with APP and BACE-1: Over-expression.The Effects of Arsenic Exposure on Neurological and Cognitive Dysfunction in Human and Rodent Studies: A ReviewEndocrine active metals, prenatal stress and enhanced neurobehavioral disruption.Effects of developmental arsenite exposure on hippocampal synapses in mouse offspring.Arsenic exposure during embryonic development alters the expression of the long noncoding RNA growth arrest specific-5 (Gas5) in a sex-dependent manner.Arsenic induces members of the mmu-miR-466-669 cluster which reduces NeuroD1 expression.Sex-Dependent Effects of the Histone Deacetylase Inhibitor, Sodium Valproate, on Reversal Learning After Developmental Arsenic Exposure.
P2860
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P2860
Moderate perinatal arsenic exposure alters neuroendocrine markers associated with depression and increases depressive-like behaviors in adult mouse offspring.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Moderate perinatal arsenic exp ...... iors in adult mouse offspring.
@en
type
label
Moderate perinatal arsenic exp ...... iors in adult mouse offspring.
@en
prefLabel
Moderate perinatal arsenic exp ...... iors in adult mouse offspring.
@en
P2093
P2860
P1433
P1476
Moderate perinatal arsenic exp ...... iors in adult mouse offspring.
@en
P2093
Andrea M Allan
Angela Bell
Bethany L Kolb
Daniel D Savage
Ebany J Martinez
P2860
P304
P356
10.1016/J.NEURO.2008.05.004
P577
2008-05-21T00:00:00Z