about
The acute phase protein Serum Amyloid A induces lipolysis and inflammation in human adipocytes through distinct pathwaysPropofol infusion syndrome: a structured review of experimental studies and 153 published case reportsThe interplay of autoimmunity and insulin resistance in type 1 diabetesCritical illness induces nutrient-independent adipogenesis and accumulation of alternatively activated tissue macrophagesSevere burn and disuse in the rat independently adversely impact body composition and adipokinesExtent and magnitude of catecholamine surge in pediatric burned patients.iNOS inhibitor, L-NIL, reverses burn-induced glycogen synthase kinase-3β activation in skeletal muscle of rats.Animal models in burn research.Age and tissue specific differences in the development of acute insulin resistance following injuryPredictors of insulin resistance in pediatric burn injury survivors 24 to 36 months postburn.Leukocyte infiltration and activation of the NLRP3 inflammasome in white adipose tissue following thermal injuryα-Tocopherol adipose tissue stores are depleted after burn injury in pediatric patients.Parenteral nutrition in the critically ill patient.Etiology of insulin resistance in youth with type 2 diabetesRole of inhibitory κB kinase and c-Jun NH2-terminal kinase in the development of hepatic insulin resistance in critical illness diabetesNovel mitochondria-targeted antioxidant peptide ameliorates burn-induced apoptosis and endoplasmic reticulum stress in the skeletal muscle of miceRole of tissue macrophages in the development of critical illness diabetesIn situ metabolic flux analysis to quantify the liver metabolic response to experimental burn injury.iNOS as a Driver of Inflammation and Apoptosis in Mouse Skeletal Muscle after Burn Injury: Possible Involvement of Sirt1 S-Nitrosylation-Mediated Acetylation of p65 NF-κB and p53Postburn Hypermetabolism: Past, Present, and Future.Mitochondrial function in skeletal muscle of patients with protracted critical illness and ICU-acquired weakness.Association of postburn fatty acids and triglycerides with clinical outcome in severely burned childrenN-3 Polyunsaturated Fatty Acids Improve Liver Lipid Oxidation-Related Enzyme Levels and Increased the Peroxisome Proliferator-Activated Receptor α Expression Level in Mice Subjected to Hemorrhagic Shock/Resuscitation.Burned Adults Develop Profound Glucose Intolerance.Nutritional Alterations Associated with Neurological and Neurosurgical Diseases.Abnormal insulin sensitivity persists up to three years in pediatric patients post-burnAdipose inflammation and macrophage infiltration after binge ethanol and burn injuryStable isotope-labeled tracers for the investigation of fatty acid and triglyceride metabolism in humans in vivoImpaired glucose tolerance in pediatric burn patients at discharge from the acute hospital stay.Lipidomic analysis enables prediction of clinical outcomes in burn patientsDevelopment of Metabolic Indicators of Burn Injury: Very Low Density Lipoprotein (VLDL) and Acetoacetate Are Highly Correlated to Severity of Burn Injury in Rats.Role of the PPAR-α agonist fenofibrate in severe pediatric burn.Role of protein farnesylation in burn-induced metabolic derangements and insulin resistance in mouse skeletal muscle.Mechanisms of burn-induced impairment in gastric slow waves and emptying in rats.Skeletal muscle mitochondria exhibit decreased pyruvate oxidation capacity and increased ROS emission during surgery-induced acute insulin resistance.Spatially discordant alternans in cardiomyocyte monolayers.Mitochondrial Function in an In Vitro Model of Skeletal Muscle of Patients With Protracted Critical Illness and Intensive Care Unit-Acquired Weakness.Percutaneous muscle biopsy-induced tissue injury causes local endoplasmic reticulum stress.
P2860
Q24317321-042B6138-B535-4A31-B552-5DAC89F1B3E5Q26777577-BB48261B-9988-44CE-85E1-90215ABF45CCQ26823862-551F15E2-B26E-4586-BB41-CA2FF535E3E4Q33751235-51BA61D3-D8AC-47E8-B744-64628D7274B7Q33753209-629B1C91-4002-4733-84CD-E2D4816D9E22Q33807436-1541D143-89FF-4327-AFDD-7F6792E9CF6CQ33878118-1FADFA4E-F9AD-4A4F-94F8-5B9690C6A318Q34051592-51399AEB-FF34-4B59-A667-2B276B8D6ECFQ34089832-C0296231-D7BC-46D8-88FF-E45460AC0593Q34173618-EEDC92C4-6692-41E9-91E5-C0EEB1597A82Q34200054-63B1C84F-309E-4963-AFBA-3C5A5899D247Q34308084-631D3AB9-DCC5-49BF-83C4-CE2BE5CA0B86Q34723274-41294701-DD2A-4045-839E-16C64D9C70F5Q34974105-5042FD53-B565-452C-B3B8-5B984F88261EQ35216722-9AA40A3F-A4E8-4877-A2F0-7F5BA58F80A3Q35591278-E64C1D7F-962E-4BB9-BEAC-C5CA0897DA5EQ35612847-CE758A4C-5008-4863-92F7-8C29E5515801Q35751814-4E6EBEF0-622B-4EB5-B293-95E7669CA730Q36252884-544C5419-72E4-4F97-BAF4-35E35D751485Q36405112-962B5EFA-EAD3-4C8F-BF43-7BFC6BF7C3CEQ36424753-30908DD6-7B8A-452F-98AE-1769B3CECA31Q36508908-7F99CE84-2C80-44D3-BFA2-BB4E62DC4B23Q36846386-565291F2-3A83-4D20-8144-C6CEE792A76BQ36906032-D007FBA5-16E1-41F5-B035-C4445421B963Q37129382-03432E19-BE98-4B99-A941-EB3FA4EEF538Q37198029-C178999C-85EF-4832-8F6A-1FCE96C5D6FEQ37297169-E6C61846-2118-4533-A084-F0E09B4301B3Q37379249-90152A61-8EE6-4C6E-B652-C2D35B49F012Q37447991-E0F46F35-57B3-4866-8372-D1CEEFE408EAQ37508648-A1613FEB-22DE-4580-82DE-AEB80D8C9E99Q37512619-125A1090-36A5-4461-9A7C-37FF5B571CFFQ37973966-AE543F49-3189-4876-90CD-4126A009E968Q41574482-1926F775-5440-4859-BE08-80B6E5D0EF99Q43081564-E9D64DA9-D1B3-4FFD-A0F7-BA7F6F37883DQ46774268-3314B209-5D37-4630-ADB6-7E464CC469B6Q46788961-57B16EEA-537E-4AFC-91C6-AAF1466512E7Q47415017-28CC8B82-7DE0-4F07-99C6-A77B3DDB0799Q55023648-1721B835-C929-4642-929B-CEF3B452383B
P2860
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Postburn trauma insulin resistance and fat metabolism.
@en
type
label
Postburn trauma insulin resistance and fat metabolism.
@en
prefLabel
Postburn trauma insulin resistance and fat metabolism.
@en
P2860
P1476
Postburn trauma insulin resistance and fat metabolism.
@en
P2093
Melanie G Cree
Robert R Wolfe
P2860
P356
10.1152/AJPENDO.00562.2007
P577
2007-10-23T00:00:00Z