TLR-2/TLR-4 TREM-1 signaling pathway is dispensable in inflammatory myeloid cells during sterile kidney injury. - Wikidata - wikimedia - TriplyDB

TLR-2/TLR-4 TREM-1 signaling pathway is dispensable in inflammatory myeloid cells during sterile kidney injury.

TLR-2/TLR-4 TREM-1 signaling pathway is dispensable in inflammatory myeloid cells during sterile kidney injury.

http://www.wikidata.org/entity/Q36981043

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The FOXD1 lineage of kidney perivascular cells and myofibroblasts: functions and responses to injury How the Innate Immune System Senses Trouble and Causes Trouble Cellular and molecular mechanisms in kidney fibrosis.CYR61 (CCN1) overexpression induces lung injury in mice Deficient Autophagy Results in Mitochondrial Dysfunction and FSGS.Dermal γδ T-Cells Can Be Activated by Mitochondrial Damage-Associated Molecular Patterns.TGF-β1 Upregulates the Expression of Triggering Receptor Expressed on Myeloid Cells 1 in Murine Lungs.CD103+ Dendritic Cells Elicit CD8+ T Cell Responses to Accelerate Kidney Injury in Adriamycin Nephropathy.MicroRNAs as novel therapeutic targets to treat kidney injury and fibrosis.Role of lung pericytes and resident fibroblasts in the pathogenesis of pulmonary fibrosis.Pericyte MyD88 and IRAK4 control inflammatory and fibrotic responses to tissue injury.Macrophage-mediated injury and repair after ischemic kidney injury.Renal erythropoietin-producing cells in health and disease.Extracellular nucleotides from dying cells act as molecular signals to promote wound repair in renal tubular injury.Identification of a Novel Splice Variant Isoform of TREM-1 in Human Neutrophil Granules.Effect of TREM-1 blockade and single nucleotide variants in experimental renal injury and kidney transplantation.Maintenance of vascular integrity by pericytes is essential for normal kidney function.sTREM-1 in patients with chronic kidney disease on hemodialysis.Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development.Presepsin: A potential biomarker of PJI? A comparative analysis with known and new infection biomarkers.Immunomodulatory Molecule IRAK-M Balances Macrophage Polarization and Determines Macrophage Responses during Renal Fibrosis.What can target kidney fibrosis?

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P2860

TLR-2/TLR-4 TREM-1 signaling pathway is dispensable in inflammatory myeloid cells during sterile kidney injury.

http://www.wikidata.org/entity/Q36981043

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

TLR-2/TLR-4 TREM-1 signaling p ...... during sterile kidney injury.

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type

label

TLR-2/TLR-4 TREM-1 signaling p ...... during sterile kidney injury.

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prefLabel

TLR-2/TLR-4 TREM-1 signaling p ...... during sterile kidney injury.

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JOURNAL.PONE.0068640

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P1476

TLR-2/TLR-4 TREM-1 signaling p ...... s during sterile kidney injury

@en

P2093

Akio Kobayashi

http://www.w3.org/2001/XMLSchema#string

Gabriela Campanholle

http://www.w3.org/2001/XMLSchema#string

Jay W Heinecke

http://www.w3.org/2001/XMLSchema#string

Jeremy S Duffield

http://www.w3.org/2001/XMLSchema#string

Jessica A Hamerman

http://www.w3.org/2001/XMLSchema#string

Kristen Mittelsteadt

http://www.w3.org/2001/XMLSchema#string

P2860

Cutting edge: inflammatory responses can be triggered by TREM-1, a novel receptor expressed on neutrophils and monocytes

Signaling danger: toll-like receptors and their potential roles in kidney disease

The renal mononuclear phagocytic system

A role for triggering receptor expressed on myeloid cells-1 in host defense during the early-induced and adaptive phases of the immune response

TREM-1 amplifies inflammation and is a crucial mediator of septic shock

Transepithelial migration of neutrophils into the lung requires TREM-1

Mincle is an ITAM-coupled activating receptor that senses damaged cells

Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells

Cutting edge: heat shock protein 60 is a putative endogenous ligand of the toll-like receptor-4 complex

Characterization of TREM-3, an activating receptor on mouse macrophages: definition of a family of single Ig domain receptors on mouse chromosome 17

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e68640

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scholarly article

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10.1371/JOURNAL.PONE.0068640

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7

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8

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Shuei-Liong Lin

William A. Altemeier

Shunsaku Nakagawa

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2013-07-03T00:00:00Z

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248398535

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23844229

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Toll-like receptor

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3700949

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