about
Proinflammatory responses by glycosylphosphatidylinositols (GPIs) of Plasmodium falciparum are mainly mediated through the recognition of TLR2/TLR1Tissue signatures influence the activation of intrahepatic CD8(+) T cells against malaria sporozoitesInsufficiently defined genetic background confounds phenotypes in transgenic studies as exemplified by malaria infection in Tlr9 knockout miceStructure and dynamic behavior of Toll-like receptor 2 subfamily triggered by malarial glycosylphosphatidylinositols of Plasmodium falciparumA distinct peripheral blood monocyte phenotype is associated with parasite inhibitory activity in acute uncomplicated Plasmodium falciparum malaria.The role of lipopeptidophosphoglycan in the immune response to Entamoeba histolytica.Interaction between Plasmodium Glycosylphosphatidylinositol and the Host Protein Moesin Has No Implication in Malaria Pathology.CD36 deficiency attenuates experimental mycobacterial infection.CD36 and TLR interactions in inflammation and phagocytosis: implications for malaria.Comparison of Plasmodium berghei challenge models for the evaluation of pre-erythrocytic malaria vaccines and their effect on perceived vaccine efficacy.Antitumor effect of malaria parasite infection in a murine Lewis lung cancer model through induction of innate and adaptive immunity.Neutralization of malaria glycosylphosphatidylinositol in vitro by serum IgG from malaria-exposed individuals.Insulin-like peptides in the mosquito Anopheles stephensi: identification and expression in response to diet and infection with Plasmodium falciparum.Toll-Like receptor 2 (TLR2) and TLR9 play opposing roles in host innate immunity against Salmonella enterica serovar Typhimurium infection.Reorienting our view of particle-based adjuvants for subunit vaccines.Mutations of complement lectin pathway genes MBL2 and MASP2 associated with placental malariaThe iron link between malaria and invasive non-typhoid Salmonella infections.The Role of TLR2 in Infection and ImmunityHuman C1-Inhibitor Suppresses Malaria Parasite Invasion and Cytoadhesion via Binding to Parasite Glycosylphosphatidylinositol and Host Cell Receptors.Inflammatory cytokine and humoral responses to Plasmodium falciparum glycosylphosphatidylinositols correlates with malaria immunity and pathogenesis.Angiopoietin-2 is associated with decreased endothelial nitric oxide and poor clinical outcome in severe falciparum malaria.Malaria modifies neonatal and early-life toll-like receptor cytokine responses.A Plasmodium yoelii soluble factor inhibits the phenotypic maturation of dendritic cells.Cellular and humoral immune responses against the Plasmodium vivax MSP-1₁₉ malaria vaccine candidate in individuals living in an endemic area in north-eastern Amazon region of Brazil.Toward automated oligosaccharide synthesis.Protective immunity against malaria after vaccination.Do you see what I see: Recognition of protozoan parasites by Toll-like receptors.The immunological balance between host and parasite in malaria.Dynamic interactions of Plasmodium spp. with vascular endothelium.Genetic predisposition of variants in TLR2 and its co-receptors to severe malaria in Odisha, India.Association of TLR variants with susceptibility to Plasmodium vivax malaria and parasitemia in the Amazon region of Brazil.Signaling Strategies of Malaria Parasite for Its Survival, Proliferation, and Infection during Erythrocytic Stage.Blood-stage malaria of Plasmodium chabaudi induces differential Tlr expression in the liver of susceptible and vaccination-protected Balb/c mice.Cytokines and Chemokines in Cerebral Malaria Pathogenesis.Peroxisome Proliferator-Activator Receptor γ: A Link between Macrophage CD36 and Inflammation in Malaria Infection.AID expression in peripheral blood of children living in a malaria holoendemic region is associated with changes in B cell subsets and Epstein-Barr virus.Monocyte dysregulation and systemic inflammation during pediatric falciparum malaria.TLR2-/- Mice Display Decreased Severity of Giardiasis via Enhanced Proinflammatory Cytokines Production Dependent on AKT Signal Pathway.Alpha-tocopherol transfer protein gene inhibition enhances the acquired immune response during malaria infection in mice.Polymorphisms and expression of TLR4 and 9 in malaria in two ethnic groups of Assam, northeast India.
P2860
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P2860
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
TLR-mediated cell signaling by malaria GPIs.
@en
type
label
TLR-mediated cell signaling by malaria GPIs.
@en
prefLabel
TLR-mediated cell signaling by malaria GPIs.
@en
P356
P1476
TLR-mediated cell signaling by malaria GPIs.
@en
P2093
D Channe Gowda
P304
P356
10.1016/J.PT.2007.09.003
P577
2007-11-05T00:00:00Z