Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice. - Wikidata - wikimedia - TriplyDB

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

http://www.wikidata.org/entity/Q37000865

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Thermoneutral housing temperature regulates T-regulatory cell function and inhibits ovabumin-induced asthma development in mice

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Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

http://www.wikidata.org/entity/Q37000865

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 14 December 2015

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@en

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@nl

type

label

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@en

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@nl

prefLabel

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@en

Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@nl

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Journal of Endocrinology

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Minimal impact of age and housing temperature on the metabolic phenotype of Acc2-/- mice.

@en

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Amanda E Brandon

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Edward W Kraegen

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Ella Stuart

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Gregory J Cooney

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Simon J Leslie

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P2860

Hypoxanthine-guanine phosophoribosyltransferase (HPRT) deficiency: Lesch-Nyhan syndrome

Gene knockout of Acc2 has little effect on body weight, fat mass, or food intake

Acetyl-CoA carboxylase 2 mutant mice are protected against obesity and diabetes induced by high-fat/high-carbohydrate diets

The glucose fatty-acid cycle. Its role in insulin sensitivity and the metabolic disturbances of diabetes mellitus

Continuous fatty acid oxidation and reduced fat storage in mice lacking acetyl-CoA carboxylase 2

Acute or chronic upregulation of mitochondrial fatty acid oxidation has no net effect on whole-body energy expenditure or adiposity.

Inducible nitric oxide synthase induction underlies lipid-induced hepatic insulin resistance in mice: potential role of tyrosine nitration of insulin signaling proteins

Disruption of thyroid hormone activation in type 2 deiodinase knockout mice causes obesity with glucose intolerance and liver steatosis only at thermoneutrality

Acetyl-CoA carboxylases 1 and 2 show distinct expression patterns in rats and humans and alterations in obesity and diabetes.

Overexpression of SIRT1 in rat skeletal muscle does not alter glucose induced insulin resistance

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127-134

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scholarly article

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10.1530/JOE-15-0444

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3

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