Triptolide induces the expression of miR-142-3p: a negative regulator of heat shock protein 70 and pancreatic cancer cell proliferation - Wikidata - wikimedia - TriplyDB

Triptolide induces the expression of miR-142-3p: a negative regulator of heat shock protein 70 and pancreatic cancer cell proliferation

Triptolide induces the expression of miR-142-3p: a negative regulator of heat shock protein 70 and pancreatic cancer cell proliferation

http://www.wikidata.org/entity/Q37001384

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Molecular targets for the treatment of pancreatic cancer: Clinical and experimental studies Emerging roles of microRNAs in pancreatic cancer diagnosis, therapy and prognosis (Review)MicroRNAs in Kidney Transplantation: Living up to Their Expectations?The human HSP70 family of chaperones: where do we stand?Triptolide activates unfolded protein response leading to chronic ER stress in pancreatic cancer cells.MicroRNAs as emerging biomarkers and therapeutic targets for pancreatic cancer Regulation of microRNAs by natural agents: new strategies in cancer therapies.Learning regulatory programs by threshold SVD regression.Phosphonooxymethyl Prodrug of Triptolide: Synthesis, Physicochemical Characterization, and Efficacy in Human Colon Adenocarcinoma and Ovarian Cancer Xenografts.The Role of Nutraceuticals in Pancreatic Cancer Prevention and Therapy: Targeting Cellular Signaling, MicroRNAs, and Epigenome.Primary and liver metastasis-derived cell lines from KrasG12D; Trp53R172H; Pdx-1 Cre animals undergo apoptosis in response to triptolide.Triptolide and its prodrug minnelide suppress Hsp70 and inhibit in vivo growth in a xenograft model of mesothelioma.Triptolide Attenuates Podocyte Injury by Regulating Expression of miRNA-344b-3p and miRNA-30b-3p in Rats with Adriamycin-Induced Nephropathy Current Perspectives on Epigenetic Modifications by Dietary Chemopreventive and Herbal Phytochemicals MiR-142-3p functions as a potential tumor suppressor directly targeting HMGB1 in non-small-cell lung carcinoma Mining for Candidate Genes Related to Pancreatic Cancer Using Protein-Protein Interactions and a Shortest Path Approach.Targeting HSF1 disrupts HSP90 chaperone function in chronic lymphocytic leukemia.Efficient detection of human circulating tumor cells without significant production of false-positive cells by a novel conditionally replicating adenovirus.Toward targeted therapy in chemotherapy-resistant pancreatic cancer with a smart triptolide nanomedicine.Diagnostic Value of Cell-free Circulating MicroRNAs for Obesity and Type 2 Diabetes: A Meta-analysis.Alpinumisoflavone induces apoptosis in esophageal squamous cell carcinoma by modulating miR-370/PIM1 signaling.Triptolide inhibits cell growth and GRP78 protein expression but induces cell apoptosis in original and radioresistant NPC cells microRNA-142 is upregulated by tumor necrosis factor-alpha and triggers apoptosis in human gingival epithelial cells by repressing BACH2 expression Radiation induces premature chromatid separation via the miR-142-3p/Bod1 pathway in carcinoma cells MicroRNAs in Pancreatic Cancer: Involvement in Carcinogenesis and Potential Use for Diagnosis and Prognosis.Targeting of Heat Shock Protein HSPA6 (HSP70B') to the Periphery of Nuclear Speckles is Disrupted by a Transcription Inhibitor Following Thermal Stress in Human Neuronal Cells.miR-142-3p inhibits cancer cell proliferation by targeting CDC25C.MicroRNA-142-3p and microRNA-142-5p are downregulated in hepatocellular carcinoma and exhibit synergistic effects on cell motility.The long non-coding RNA TP73-AS1 interacted with miR-142 to modulate brain glioma growth through HMGB1/RAGE pathway.Triptolide synergistically enhances temozolomide-induced apoptosis and potentiates inhibition of NF-κB signaling in glioma initiating cells.Anticancer Natural Compounds as Epigenetic Modulators of Gene Expression Effect of the Natural Product Triptolide on Pancreatic Cancer: A Systematic Review of Preclinical Studies MicroRNA-337 inhibits cell proliferation and invasion of cervical cancer through directly targeting specificity protein 1.MicroRNA-142-3p inhibits cell proliferation and invasion of cervical cancer cells by targeting FZD7.MiR-142-3p is downregulated in aggressive p53 mutant mouse models of pancreatic ductal adenocarcinoma by hypermethylation of its locus.Exosomal Chaperones and miRNAs in Gliomagenesis: State-of-Art and Theranostics Perspectives Nano and Microsensors for Mammalian Cell Studies

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Triptolide induces the expression of miR-142-3p: a negative regulator of heat shock protein 70 and pancreatic cancer cell proliferation

http://www.wikidata.org/entity/Q37001384

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on May 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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name

Triptolide induces the express ...... atic cancer cell proliferation

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Triptolide induces the express ...... tic cancer cell proliferation.

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Triptolide induces the express ...... atic cancer cell proliferation

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Triptolide induces the express ...... tic cancer cell proliferation.

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Triptolide induces the express ...... atic cancer cell proliferation

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Triptolide induces the express ...... tic cancer cell proliferation.

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Molecular Cancer Therapeutics

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Triptolide induces the express ...... atic cancer cell proliferation

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Aaron Sarver

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Ashok K Saluja

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Nameeta Mujumdar

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Selwyn Vickers

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Sulagna Banerjee

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Tiffany N MacKenzie

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Veena Sangwan

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P2860

Coordinated post-transcriptional regulation of Hsp70.3 gene expression by microRNA and alternative polyadenylation

Tumor protein 53-induced nuclear protein 1 expression is repressed by miR-155, and its restoration inhibits pancreatic tumor development

Cancer statistics, 2010

Suppression of expression of heat shock protein 70 by gefitinib and its contribution to pulmonary fibrosis

Enhancing chemosensitivity to gemcitabine via RNA interference targeting the catalytic subunits of protein kinase CK2 in human pancreatic cancer cells

Heat shock protein 70 inhibits apoptosis in cancer cells through simultaneous and independent mechanisms

Neuronal markers are expressed in human gliomas and NSE knockdown sensitizes glioblastoma cells to radiotherapy and temozolomide

Targeting microRNAs in cancer: rationale, strategies and challenges.

ER stress negatively modulates the expression of the miR-199a/214 cluster to regulates tumor survival and progression in human hepatocellular cancer.

Expression profiling identifies microRNA signature in pancreatic cancer.

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1266-1275

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scholarly article

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10.1158/1535-7163.MCT-12-1231

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7

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12

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Subbaya Subramanian

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2013-05-01T00:00:00Z

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23635652

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3707985

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