TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes
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Ion Channels in the HeartCooperative endocytosis of the endosomal SNARE protein syntaxin-8 and the potassium channel TASK-1.Gain-of-function mutation in TASK-4 channels and severe cardiac conduction disorder.Modulation of K2P 2.1 and K2P 10.1 K(+) channel sensitivity to carvedilol by alternative mRNA translation initiation.TASK-1 current is inhibited by phosphorylation during human and canine chronic atrial fibrillation.Breathing Stimulant Compounds Inhibit TASK-3 Potassium Channel Function Likely by Binding at a Common Site in the Channel Pore.Ability to induce atrial fibrillation in the peri-operative period is associated with phosphorylation-dependent inhibition of TWIK protein-related acid-sensitive potassium channel 1 (TASK-1)PITX2 Modulates Atrial Membrane Potential and the Antiarrhythmic Effects of Sodium-Channel Blockers.New directions in antiarrhythmic drug therapy for atrial fibrillation.Cardiac potassium channel subtypes: new roles in repolarization and arrhythmia.The role of acid-sensitive two-pore domain potassium channels in cardiac electrophysiology: focus on arrhythmias.Contribution of two-pore K+ channels to cardiac ventricular action potential revealed using human iPSC-derived cardiomyocytes.Novel ion channel targets in atrial fibrillation.Anti-arrhythmic strategies for atrial fibrillation: The role of computational modeling in discovery, development, and optimization.Functional and molecular identification of a TASK-1 potassium channel regulating chloride secretion through CFTR channels in the shark rectal gland: implications for cystic fibrosis.Inhibition of a TREK-like K+ channel current by noradrenaline requires both β1- and β2-adrenoceptors in rat atrial myocytes.pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.Kv1.5 blockers preferentially inhibit TASK-1 channels: TASK-1 as a target against atrial fibrillation and obstructive sleep apnea?Cardiac Potassium Channels: Physiological Insights for Targeted Therapy.Novel electrophysiological properties of dronedarone: inhibition of human cardiac two-pore-domain potassium (K2P) channels.The inhibition of the potassium channel TASK-1 in rat cardiac muscle by endothelin-1 is mediated by phospholipase C.The potassium current carried by TREK-1 channels in rat cardiac ventricular muscle.Computational Modeling of Electrophysiology and Pharmacotherapy of Atrial Fibrillation: Recent Advances and Future ChallengesNeurokinin-3 receptor activation selectively prolongs atrial refractoriness by inhibition of a background K channel
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P2860
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 December 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes
@en
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes.
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type
label
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes
@en
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes.
@nl
prefLabel
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes
@en
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes.
@nl
P2093
P2860
P356
P1476
TASK-1 channels may modulate action potential duration of human atrial cardiomyocytes
@en
P2093
Caroline Rolfes
Günter Schlichthörl
Hinnerk Wulf
Jürgen Daut
Marylou Zuzarte
Michael F Netter
Rainer Moosdorf
Susanne Rinné
Sven H Limberg
Timon Vassiliou
P2860
P304
P356
10.1159/000335757
P577
2011-12-14T00:00:00Z