HIV-1 Transgenic Rat Prefrontal Cortex Hyper-Excitability is Enhanced by Cocaine Self-Administration. - Wikidata - wikimedia - TriplyDB

HIV-1 Transgenic Rat Prefrontal Cortex Hyper-Excitability is Enhanced by Cocaine Self-Administration.

HIV-1 Transgenic Rat Prefrontal Cortex Hyper-Excitability is Enhanced by Cocaine Self-Administration.

http://www.wikidata.org/entity/Q37006276

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HIV Tat excites D1 receptor-like expressing neurons from rat nucleus accumbens.Combined chronic blockade of hyper-active L-type calcium channels and NMDA receptors ameliorates HIV-1 associated hyper-excitability of mPFC pyramidal neurons.Colon dysregulation in methamphetamine self-administering HIV-1 transgenic rats.Aging alters voltage-gated calcium channels in prefrontal cortex pyramidal neurons in the HIV brain.Impact on Cortical Function of Cocaine Abuse Co-Occurring with HIV.Enhanced neuronal and blunted hemodynamic reactivity to cocaine in the prefrontal cortex following extended cocaine access: optical imaging study in anesthetized rats.HIV-1 proteins dysregulate motivational processes and dopamine circuitry.

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HIV-1 Transgenic Rat Prefrontal Cortex Hyper-Excitability is Enhanced by Cocaine Self-Administration.

http://www.wikidata.org/entity/Q37006276

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 December 2015

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

@en

HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

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type

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HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

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HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

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HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

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HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

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Neuropsychopharmacology

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HIV-1 Transgenic Rat Prefronta ...... y Cocaine Self-Administration.

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Lihua Chen

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T Celeste Napier

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Wesley N Wayman

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Xiu-Ti Hu

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P2860

HIV-associated neurocognitive disorders before and during the era of combination antiretroviral therapy: differences in rates, nature, and predictors

Cortical consequences of HIV-1 Tat exposure in rats are enhanced by chronic cocaine

The neuropathogenesis of AIDS

Synergistic neurotoxicity by human immunodeficiency virus proteins Tat and gp120: protection by memantine

Regulation of synaptic efficacy by coincidence of postsynaptic APs and EPSPs

Drug addiction and its underlying neurobiological basis: neuroimaging evidence for the involvement of the frontal cortex.

Dose escalation and dose preference in extended-access heroin self-administration in Lewis and Fischer rats

Repeated cocaine treatment enhances HIV-1 Tat-induced cortical excitability via over-activation of L-type calcium channels.

An HIV-1 transgenic rat that develops HIV-related pathology and immunologic dysfunction.

Thinning of the cerebral cortex visualized in HIV/AIDS reflects CD4+ T lymphocyte decline.

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1965-1973

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10.1038/NPP.2015.366

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