Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance. - Wikidata - wikimedia - TriplyDB

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

http://www.wikidata.org/entity/Q37010103

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Matrix metalloproteinase-2 polymorphisms and clinical outcome of Chinese patients with nonsmall cell lung cancer treated with first-line, platinum-based chemotherapy.Tissue inhibitor of metalloproteinase-3 (TIMP-3) regulates hematopoiesis and bone formation in vivo.TIMP-1 deficiency subverts cell-cycle dynamics in murine long-term HSCs.

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Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

http://www.wikidata.org/entity/Q37010103

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 19 May 2008

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

@en

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

@nl

type

label

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

@en

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

@nl

prefLabel

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

@en

Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance.

@nl

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J.BCMD.2008.03.005

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Blood Cells, Molecules and Diseases

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Hematopoiesis in mice is extremely resilient to wide variation in TIMP/MMP balance

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Eleonora Haviernikova

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Kevin D Bunting

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Maria T Diaz

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Peter Haviernik

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William Tse

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P2860

Identification of CD63 as a tissue inhibitor of metalloproteinase-1 interacting cell surface protein

Recruitment of stem and progenitor cells from the bone marrow niche requires MMP-9 mediated release of kit-ligand

Tissue inhibitors of metalloproteinases: evolution, structure and function

Matrix metalloproteinases

Gelatinase B functions as regulator and effector in leukocyte biology

Neutrophil gelatinase B and chemokines in leukocytosis and stem cell mobilization

Rescue of mammary epithelial cell apoptosis and entactin degradation by a tissue inhibitor of metalloproteinases-1 transgene.

The production of tissue inhibitors of metalloproteinases (TIMPs) in megakaryopoiesis: possible role of platelet- and megakaryocyte-derived TIMPs in bone marrow fibrosis.

Receptors for interleukin (IL)-10 and IL-6-type cytokines use similar signaling mechanisms for inducing transcription through IL-6 response elements.

IL-10 inhibits metalloproteinase and stimulates TIMP-1 production in human mononuclear phagocytes

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179-187

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scholarly article

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10.1016/J.BCMD.2008.03.005

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2

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