Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth. - Wikidata - wikimedia - TriplyDB

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

http://www.wikidata.org/entity/Q37011837

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RNA-binding proteins, neural development and the addictions Competing Interactions of RNA-Binding Proteins, MicroRNAs, and Their Targets Control Neuronal Development and Function Local translation in neuronal processes--in vivo tests of a "heretical hypothesis"The RNA Binding Protein Igf2bp1 Is Required for Zebrafish RGC Axon Outgrowth In Vivo Macrophage repolarization with targeted alginate nanoparticles containing IL-10 plasmid DNA for the treatment of experimental arthritis Growth control mechanisms in neuronal regeneration A HuD-ZBP1 ribonucleoprotein complex localizes GAP-43 mRNA into axons through its 3' untranslated region AU-rich regulatory element.Prenatal deletion of the RNA-binding protein HuD disrupts postnatal cortical circuit maturation and behavior Dysregulation of the axonal trafficking of nuclear-encoded mitochondrial mRNA alters neuronal mitochondrial activity and mouse behavior Messenger RNAs localized to distal projections of human stem cell derived neurons.ZBP1 phosphorylation at serine 181 regulates its dendritic transport and the development of dendritic trees of hippocampal neurons.Luteal activity of pregnant rats with hypo-and hyperthyroidism.Syntaxin13 expression is regulated by mammalian target of rapamycin (mTOR) in injured neurons to promote axon regeneration Cell biology in neuroscience: Cellular and molecular mechanisms underlying axon formation, growth, and branching.Sensing nerve injury at the axonal ER: activated Luman/CREB3 serves as a novel axonally synthesized retrograde regeneration signal.Aberrant RNA homeostasis in amyotrophic lateral sclerosis: potential for new therapeutic targets?Axonal amphoterin mRNA is regulated by translational control and enhances axon outgrowth mRNAs and Protein Synthetic Machinery Localize into Regenerating Spinal Cord Axons When They Are Provided a Substrate That Supports Growth.Local axonal protection by WldS as revealed by conditional regulation of protein stability.Filamin A is required in injured axons for HDAC5 activity and axon regeneration.Old dogs with new tricks: intra-axonal translation of nuclear proteins.Molecular and Cellular Mechanisms of Axonal Regeneration After Spinal Cord Injury.hnRNP-Q1 represses nascent axon growth in cortical neurons by inhibiting Gap-43 mRNA translation Deficiency of the Survival of Motor Neuron Protein Impairs mRNA Localization and Local Translation in the Growth Cone of Motor Neurons.Dynamic Changes in Local Protein Synthetic Machinery in Regenerating Central Nervous System Axons after Spinal Cord Injury.Isolation and analyses of axonal ribonucleoprotein complexes Methods to study maternal regulation of germ cell specification in zebrafish KSRP modulation of GAP-43 mRNA stability restricts axonal outgrowth in embryonic hippocampal neurons.Dynamics of survival of motor neuron (SMN) protein interaction with the mRNA-binding protein IMP1 facilitates its trafficking into motor neuron axons.Molecular determinants of the axonal mRNA transcriptome.Neural Progenitor Cells Promote Axonal Growth and Alter Axonal mRNA Localization in Adult Neurons.Regulation of mRNA transport, localization and translation in the nervous system of mammals (Review).RNA-binding proteins and translational regulation in axons and growth cones.Mechanisms for axon maintenance and plasticity in motoneurons: alterations in motoneuron disease.Cell intrinsic control of axon regeneration.Involvement of Rho-family GTPases in axon branching.Membrane turnover and receptor trafficking in regenerating axons.Advances in understanding the role of disease-associated proteins in spinal muscular atrophy.Intra-axonal protein synthesis in development and beyond.Differential roles of α-, β-, and γ-actin in axon growth and collateral branch formation in motoneurons.

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Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

http://www.wikidata.org/entity/Q37011837

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on February 2013

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@en

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@nl

type

label

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@en

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@nl

prefLabel

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@en

Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@nl

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JNEUROSCI.1722-12.2013

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Journal of Neuroscience

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Axonally synthesized β-actin and GAP-43 proteins support distinct modes of axonal growth.

@en

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Almudena Pacheco

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Christopher J Donnelly

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Cynthia Gomes

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Deepika Vuppalanchi

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Gianluca Gallo

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Hak Hee Kim

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Jeffery L Twiss

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Marguerite McDonald

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Michael Park

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Mirela Spillane

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P2860

Molecular composition of IMP1 ribonucleoprotein granules

GAP-43: an intrinsic determinant of neuronal development and plasticity

The cytoskeletal and signaling mechanisms of axon collateral branching

Neuronal pathfinding is abnormal in mice lacking the neuronal growth cone protein GAP-43

Intra-axonal translation and retrograde trafficking of CREB promotes neuronal survival

Translational control of ribosomal protein L4 mRNA is required for rapid neurite regeneration

An NGF-responsive element targets myo-inositol monophosphatase-1 mRNA to sympathetic neuron axons

Differential transport and local translation of cytoskeletal, injury-response, and neurodegeneration protein mRNAs in axons

Restricted morphological and behavioral abnormalities following ablation of β-actin in the brain

The insulin-like growth factor mRNA binding-protein IMP-1 and the Ras-regulatory protein G3BP associate with tau mRNA and HuD protein in differentiated P19 neuronal cells.

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3311-3322

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scholarly article

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10.1523/JNEUROSCI.1722-12.2013

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8

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33

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2013-02-01T00:00:00Z

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235682245

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23426659

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3711152

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