Osteocalcin Signaling in Myofibers Is Necessary and Sufficient for Optimum Adaptation to Exercise - Wikidata - wikimedia - TriplyDB

Osteocalcin Signaling in Myofibers Is Necessary and Sufficient for Optimum Adaptation to Exercise

Osteocalcin Signaling in Myofibers Is Necessary and Sufficient for Optimum Adaptation to Exercise

http://www.wikidata.org/entity/Q37012238

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Diabetes, bone and glucose-lowering agents: basic biology.CRISPR/Cas9 targeting of GPRC6A suppresses prostate cancer tumorigenesis in a human xenograft model Bone muscle crosstalk targets muscle regeneration pathway regulated by core circadian transcriptional repressors DEC1 and DEC2.Osteocalcin is necessary and sufficient to maintain muscle mass in older mice.GPRC6A: Jack of all metabolism (or master of none).Non-alcoholic fatty liver disease connections with fat-free tissues: A focus on bone and skeletal muscle Role of the Cytokine-like Hormone Leptin in Muscle-bone Crosstalk with Aging.Osteocalcin-dependent regulation of glucose metabolism and fertility: Skeletal implications for the development of insulin resistance.Low serum osteocalcin concentration is associated with incident type 2 diabetes mellitus in Japanese women.Functional Study of the Vitamin K Cycle Enzymes in Live Cells.The Role of the Osteocyte in Bone and Nonbone Disease.Bone Cell Bioenergetics and Skeletal Energy Homeostasis.Multifaceted interaction of bone, muscle, lifestyle interventions and metabolic and cardiovascular disease: role of osteocalcin.Osteocalcin, a bone-derived hormone with important andrological implications.Vitamin D, a modulator of musculoskeletal health in chronic kidney disease.Effect of Weight Loss, Exercise, or Both on Undercarboxylated Osteocalcin and Insulin Secretion in Frail, Obese Older Adults.Quick and inexpensive paraffin-embedding method for dynamic bone formation analyses.Recombinant Uncarboxylated Osteocalcin Per Se Enhances Mouse Skeletal Muscle Glucose Uptake in both Extensor Digitorum Longus and Soleus Muscles.Osteocalcin expressing cells from tendon sheaths in mice contribute to tendon repair by activating Hedgehog signaling.Proprotein convertase furin regulates osteocalcin and bone endocrine function.Osteocalcin in the brain: from embryonic development to age-related decline in cognition.Evaluation of Short and Long Term Cold Stress Challenge of Nerve Grow Factor, Brain-Derived Neurotrophic Factor, Osteocalcin and Oxytocin mRNA Expression in BAT, Brain, Bone and Reproductive Tissue of Male Mice Using Real-Time PCR and Linear Correla Gpr158 mediates osteocalcin's regulation of cognition.Maternal obesity increases insulin resistance, low-grade inflammation and osteochondrosis lesions in foals and yearlings until 18 months of age.Inadequate Dietary Phosphorus Levels Cause Skeletal Anomalies and Alter Osteocalcin Gene Expression in Zebrafish.Role of Inactivity in Chronic Diseases: Evolutionary Insight and Pathophysiological Mechanisms.Exercise metabolism in 2016: Health benefits of exercise - more than meets the eye!TCF7/TCF1 feedback controls osteocalcin signaling in brown adipocytes independent of canonical WNT/β-catenin pathway.The potential role of the osteopontin-osteocalcin-osteoprotegerin triad in the pathogenesis of prediabetes in humans.The Expanding Life and Functions of Osteogenic Cells: From Simple Bone-Making Cells to Multifunctional Cells and Beyond.Sclerostin and energy metabolism during physical activity.Skeletal Muscle as an Endocrine Organ: The Role of Myokines in Exercise Adaptations.Structure and Function of Bone Marrow Adipocytes.Muscle-Bone Crosstalk: Emerging Opportunities for Novel Therapeutic Approaches to Treat Musculoskeletal Pathologies.Regulation of Glucose Handling by the Skeleton: Insights From Mouse and Human Studies.Paracrine and endocrine actions of bone-the functions of secretory proteins from osteoblasts, osteocytes, and osteoclasts.Osteocalcin Ameliorates Motor Dysfunction in a 6-Hydroxydopamine-Induced Parkinson's Disease Rat Model Through AKT/GSK3β Signaling Endocrine Crosstalk Between Skeletal Muscle and the Brain mTORC Inhibitors as Broad-Spectrum Therapeutics for Age-Related Diseases Bone hormone boosts exercise

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Osteocalcin Signaling in Myofibers Is Necessary and Sufficient for Optimum Adaptation to Exercise

http://www.wikidata.org/entity/Q37012238

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on June 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Osteocalcin Signaling in Myofi ...... Optimum Adaptation to Exercise

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Osteocalcin Signaling in Myofi ...... ptimum Adaptation to Exercise.

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Osteocalcin Signaling in Myofi ...... Optimum Adaptation to Exercise

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Osteocalcin Signaling in Myofi ...... ptimum Adaptation to Exercise.

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Osteocalcin Signaling in Myofi ...... Optimum Adaptation to Exercise

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Osteocalcin Signaling in Myofi ...... ptimum Adaptation to Exercise.

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J.CMET.2016.05.004

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Cell Metabolism

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Osteocalcin Signaling in Myofi ...... Optimum Adaptation to Exercise

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Christopher Torsitano

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Cyril Confavreux

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Gerard Karsenty

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Irwin Kurland

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Jianwen Wei

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Julie A Mattison

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Justine Bacchetta

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Kathrin Laue

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Mathieu Ferron

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Michelle Puchowicz

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Endocrine regulation of energy metabolism by the skeleton

Insulin signalling and the regulation of glucose and lipid metabolism

Contraction stimulates translocation of glucose transporter GLUT4 in skeletal muscle through a mechanism distinct from that of insulin

Soluble interleukin-6 receptor triggers osteoclast formation by interleukin 6

The search for exercise factors in humans

A PGC1-α-dependent myokine that drives brown-fat-like development of white fat and thermogenesis

Genetic regulation of osteoclast development and function

Mitochondrial division is requisite to RAS-induced transformation and targeted by oncogenic MAPK pathway inhibitors.

Contraction-induced interleukin-6 gene transcription in skeletal muscle is regulated by c-Jun terminal kinase/activator protein-1

Elevated PGC-1α activity sustains mitochondrial biogenesis and muscle function without extending survival in a mouse model of inherited ALS

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1078-1092

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scholarly article

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10.1016/J.CMET.2016.05.004

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6

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23

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Alain Lacampagne

Marta Galán-Díez

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2016-06-01T00:00:00Z

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27304508

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4910629

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