Hyperglycemia-induced protein kinase C β2 activation induces diastolic cardiac dysfunction in diabetic rats by impairing caveolin-3 expression and Akt/eNOS signaling.
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Protein kinase C beta II upregulates intercellular adhesion molecule-1 via mitochondrial activation in cultured endothelial cellsN-acetylcysteine and allopurinol confer synergy in attenuating myocardial ischemia injury via restoring HIF-1α/HO-1 signaling in diabetic rats.Complex regulation of PKCβ2 and PDK-1/AKT by ROCK2 in diabetic heart.Gαq protein carboxyl terminus imitation polypeptide GCIP-27 improves cardiac function in chronic heart failure rats.Metabolic alterations induce oxidative stress in diabetic and failing hearts: different pathways, same outcome.Influence of glycemic control on gain in VO2 peak, in patients with type 2 diabetes enrolled in cardiac rehabilitation after an acute coronary syndrome. The prospective DARE studyNeuroprotective Effect of Ulinastatin on Spinal Cord Ischemia-Reperfusion Injury in Rabbits.Nitric Oxide Induces Cardiac Protection by Preventing Extracellular Matrix Degradation through the Complex Caveolin-3/EMMPRIN in Cardiac Myocytes.MicroRNA-126 contributes to Niaspan treatment induced vascular restoration after diabetic retinopathy.N-acetylcysteine attenuates myocardial dysfunction and postischemic injury by restoring caveolin-3/eNOS signaling in diabetic rats.Cox-2 Inhibition Protects against Hypoxia/Reoxygenation-Induced Cardiomyocyte Apoptosis via Akt-Dependent Enhancement of iNOS ExpressionNitroglycerine-induced nitrate tolerance compromises propofol protection of the endothelial cells against TNF-α: the role of PKC-β2 and NADPH oxidase.N-Acetylcysteine Attenuates Diabetic Myocardial Ischemia Reperfusion Injury through Inhibiting Excessive Autophagy.Niaspan inhibits diabetic retinopathy‑induced vascular inflammation by downregulating the tumor necrosis factor‑α pathway.Damaging effects of hyperglycemia on cardiovascular function: spotlight on glucose metabolic pathways.High Glucose Enhances Isoflurane-Induced Neurotoxicity by Regulating TRPC-Dependent Calcium Influx.Inhibition of PKCβ2 overexpression ameliorates myocardial ischaemia/reperfusion injury in diabetic rats via restoring caveolin-3/Akt signaling.Sarcolemmal dependence of cardiac protection and stress-resistance: roles in aged or diseased hearts.Caveolin proteins: a molecular insight into disease.Flow signaling and atherosclerosis.Macro- and microvascular endothelial dysfunction in diabetes.IGFBP-3 reduces eNOS and PKCzeta phosphorylation, leading to lowered VEGF levels.Intravenous Infusion of Dexmedetomidine Combined Isoflurane Inhalation Reduces Oxidative Stress and Potentiates Hypoxia Pulmonary Vasoconstriction during One-Lung Ventilation in PatientsThe Protective Effect of Antioxidants Consumption on Diabetes and Vascular ComplicationsMyocyte membrane and microdomain modifications in diabetes: determinants of ischemic tolerance and cardioprotection.AMPK activation by glucagon-like peptide-1 prevents NADPH oxidase activation induced by hyperglycemia in adult cardiomyocytes.Protein Kinase C β: a New Target Therapy to Prevent the Long-Term Atypical Antipsychotic-Induced Weight Gain.Blockade of PKCβ protects against remote organ injury induced by intestinal ischemia and reperfusion via a p66shc-mediated mitochondrial apoptotic pathway.Novel protective role of the circadian nuclear receptor retinoic acid-related orphan receptor-α in diabetic cardiomyopathy.The Rho kinase inhibitor, fasudil, ameliorates diabetes-induced cardiac dysfunction by improving calcium clearance and actin remodeling.Glycine Protects H9C2 Cardiomyocytes from High Glucose- and Hypoxia/Reoxygenation-Induced Injury via Inhibiting PKCβ2 Activation and Improving Mitochondrial Quality.Polydatin Protects Diabetic Heart against Ischemia-Reperfusion Injury via Notch1/Hes1-Mediated Activation of Pten/Akt Signaling.MiR-21 protected against diabetic cardiomyopathy induced diastolic dysfunction by targeting gelsolinExogenous Pancreatic Kallikrein Improves Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetes
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Hyperglycemia-induced protein kinase C β2 activation induces diastolic cardiac dysfunction in diabetic rats by impairing caveolin-3 expression and Akt/eNOS signaling.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 08 March 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Hyperglycemia-induced protein ...... ession and Akt/eNOS signaling.
@en
Hyperglycemia-induced protein ...... ession and Akt/eNOS signaling.
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type
label
Hyperglycemia-induced protein ...... ession and Akt/eNOS signaling.
@en
Hyperglycemia-induced protein ...... ession and Akt/eNOS signaling.
@nl
prefLabel
Hyperglycemia-induced protein ...... ession and Akt/eNOS signaling.
@en
Hyperglycemia-induced protein ...... ession and Akt/eNOS signaling.
@nl
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P2860
P50
P356
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P1476
Hyperglycemia-induced protein ...... ression and Akt/eNOS signaling
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Brian Rodrigues
Kwok F J Ng
Shaoqing Lei
Wayne Bond Lau
Xin-Liang Ma
Zhengyuan Xia
P2860
P304
P356
10.2337/DB12-1391
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P577
2013-03-08T00:00:00Z