Molecular mechanisms underlying a cellular analog of operant reward learning.
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More than synaptic plasticity: role of nonsynaptic plasticity in learning and memoryDrosophila FoxP mutants are deficient in operant self-learningChronic cocaine disrupts mesocortical learning mechanismsPKC in motorneurons underlies self-learning, a form of motor learning in DrosophilaCritical role for protein kinase A in the acquisition of gregarious behavior in the desert locust.Operant conditioning: a minimal components requirement in artificial spiking neurons designed for bio-inspired robot's controller.Evolutionary conservation of the signaling proteins upstream of cyclic AMP-dependent kinase and protein kinase C in gastropod mollusks.Serotonin stimulation of cAMP-dependent plasticity in Aplysia sensory neurons is mediated by calmodulin-sensitive adenylyl cyclase.Massed training-induced intermediate-term operant memory in aplysia requires protein synthesis and multiple persistent kinase cascadesPKA and PKC are required for long-term but not short-term in vivo operant memory in Aplysia.Classical conditioning analog enhanced acetylcholine responses but reduced excitability of an identified neuronAssociative learning in invertebrates.Reduced activity of protein kinase C in the frontal cortex of subjects with regressive autism: relationship with developmental abnormalitiesPKA has a critical role in synaptic delivery of GluR1- and GluR4-containing AMPARs during initial stages of acquisition of in vitro classical conditioning.The atypical protein kinase C in Aplysia can form a protein kinase M by cleavageNeural mechanisms of operant conditioning and learning-induced behavioral plasticity in Aplysia.Invertebrate learning and cognition: relating phenomena to neural substrate.The malleable brain: plasticity of neural circuits and behavior - a review from students to students.Cell-Specific PKM Isoforms Contribute to the Maintenance of Different Forms of Persistent Long-Term Synaptic Plasticity.The biology of psychology: 'Simple' conditioning?PKG-mediated MAPK signaling is necessary for long-term operant memory in Aplysia.Unique Configurations of Compression and Truncation of Neuronal Activity Underlie l-DOPA-Induced Selection of Motor Patterns in Aplysia.Modeling signal transduction in classical conditioning with network motifs.Coincidence detection in a neural correlate of classical conditioning is initiated by bidirectional 3-phosphoinositide-dependent kinase-1 signalling and modulated by adenosine receptors.Spermidine-induced improvement of memory involves a cross-talk between protein kinases C and A.Long-term sensitization training in Aplysia decreases the excitability of a decision-making neuron through a sodium-dependent mechanism.Inhibitory responses in Aplysia pleural sensory neurons act to block excitability, transmitter release, and PKC Apl II activation
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Molecular mechanisms underlying a cellular analog of operant reward learning.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on September 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Molecular mechanisms underlying a cellular analog of operant reward learning.
@en
Molecular mechanisms underlying a cellular analog of operant reward learning.
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type
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Molecular mechanisms underlying a cellular analog of operant reward learning.
@en
Molecular mechanisms underlying a cellular analog of operant reward learning.
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prefLabel
Molecular mechanisms underlying a cellular analog of operant reward learning.
@en
Molecular mechanisms underlying a cellular analog of operant reward learning.
@nl
P2093
P2860
P1433
P1476
Molecular mechanisms underlying a cellular analog of operant reward learning.
@en
P2093
Douglas A Baxter
Fred D Lorenzetti
John H Byrne
P2860
P304
P356
10.1016/J.NEURON.2008.07.019
P407
P577
2008-09-01T00:00:00Z