Comprehensive Genomic Profiling Identifies a Subset of Crizotinib-Responsive ALK-Rearranged Non-Small Cell Lung Cancer Not Detected by Fluorescence In Situ Hybridization - Wikidata - wikimedia - TriplyDB

Comprehensive Genomic Profiling Identifies a Subset of Crizotinib-Responsive ALK-Rearranged Non-Small Cell Lung Cancer Not Detected by Fluorescence In Situ Hybridization

Comprehensive Genomic Profiling Identifies a Subset of Crizotinib-Responsive ALK-Rearranged Non-Small Cell Lung Cancer Not Detected by Fluorescence In Situ Hybridization

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ALK Fusion Detection in Circulating Free DNA: Finding an Important Needle in the Haystack Post-crizotinib management of effective ceritinib therapy in a patient with ALK-positive non-small cell lung cancer.Detection of ALK fusion transcripts in FFPE lung cancer samples by NanoString technology ALK Status Assessment with Liquid Biopsies of Lung Cancer Patients.Performance of a RT-PCR Assay in Comparison to FISH and Immunohistochemistry for the Detection of ALK in Non-Small Cell Lung Cancer.ALK gene alterations in cancer: biological aspects and therapeutic implications.ALK alterations and inhibition in lung cancer.The impact of immunohistochemistry on the classification of lung tumors.Targeting ALK: Precision Medicine Takes on Drug Resistance.ALK in Non-Small Cell Lung Cancer (NSCLC) Pathobiology, Epidemiology, Detection from Tumor Tissue and Algorithm Diagnosis in a Daily Practice.Response to crizotinib in a lung adenocarcinoma patient harboring a novel SLC34A2-ROS1 fusion variant.Screening for ALK Rearrangements in Lung Cancer: Time for a New Generation of Diagnostics?Liquid biopsy in the practice of neo-oncology ALK Fusions in a Wide Variety of Tumor Types Respond to Anti-ALK Targeted Therapy.Genomic heterogeneity of ALK fusion breakpoints in non-small-cell lung cancer.Impact of EML4-ALK Variant on Resistance Mechanisms and Clinical Outcomes in ALK-Positive Lung Cancer.ALK-rearrangement in non-small-cell lung cancer (NSCLC).Clinical utility of cell-free DNA for the detection of ALK fusions and genomic mechanisms of ALK inhibitor resistance in non-small cell lung cancer.Clinical outcome study of crizotinib in immunohistochemistry-proven echinoderm microtubule-associated protein-like 4-anaplastic lymphoma kinase fusion gene among Indian patients with adenocarcinoma lung.Simultaneous detection of lung fusions using a multiplex RT-PCR next generation sequencing-based approach: a multi-institutional research study

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Comprehensive Genomic Profiling Identifies a Subset of Crizotinib-Responsive ALK-Rearranged Non-Small Cell Lung Cancer Not Detected by Fluorescence In Situ Hybridization

http://www.wikidata.org/entity/Q37017049

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 31 May 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Comprehensive Genomic Profilin ...... rescence In Situ Hybridization

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Comprehensive Genomic Profilin ...... escence In Situ Hybridization.

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Comprehensive Genomic Profilin ...... rescence In Situ Hybridization

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Comprehensive Genomic Profilin ...... escence In Situ Hybridization.

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prefLabel

Comprehensive Genomic Profilin ...... rescence In Situ Hybridization

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Comprehensive Genomic Profilin ...... escence In Situ Hybridization.

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THEONCOLOGIST.2015-0497

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Comprehensive Genomic Profilin ...... rescence In Situ Hybridization

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Alexa B Schrock

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Atul Kulkarni

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Doron Lipson

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Eric Sanford

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Heather Wakelee

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Heber MacMahon

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Jan Nowak

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Jeffrey S Ross

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Jie He

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P2860

Differential protein stability and ALK inhibitor sensitivity of EML4-ALK fusion variants.

Crizotinib versus chemotherapy in advanced ALK-positive lung cancer.

Ceritinib in ALK-rearranged non-small-cell lung cancer.

Identification of a novel HIP1-ALK fusion variant in Non-Small-Cell Lung Cancer (NSCLC) and discovery of ALK I1171 (I1171N/S) mutations in two ALK-rearranged NSCLC patients with resistance to Alectinib.

First-line crizotinib versus chemotherapy in ALK-positive lung cancer.

Acquired resistance of EGFR-mutant lung adenocarcinomas to afatinib plus cetuximab is associated with activation of mTORC1.

New targetable oncogenes in non-small-cell lung cancer.

Broad, Hybrid Capture-Based Next-Generation Sequencing Identifies Actionable Genomic Alterations in Lung Adenocarcinomas Otherwise Negative for Such Alterations by Other Genomic Testing Approaches

Tyrosine kinase gene rearrangements in epithelial malignancies.

Refining the treatment of NSCLC according to histological and molecular subtypes.

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10.1634/THEONCOLOGIST.2015-0497

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Garrett M Frampton

Sai-Hong Ignatius Ou

Juliann Chmielecki

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2016-05-31T00:00:00Z

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