Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy
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Replicate high-density rat genome oligonucleotide microarrays reveal hundreds of regulated genes in the dorsal root ganglion after peripheral nerve injuryConotoxins That Could Provide Analgesia through Voltage Gated Sodium Channel InhibitionStructure and function of μ-conotoxins, peptide-based sodium channel blockers with analgesic activityRoles of Voltage-Gated Tetrodotoxin-Sensitive Sodium Channels NaV1.3 and NaV1.7 in Diabetes and Painful Diabetic NeuropathySodium currents of large (Abeta-type) adult cutaneous afferent dorsal root ganglion neurons display rapid recovery from inactivation before and after axotomyInhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of PainExpression of background potassium channels in rat DRG is cell-specific and down-regulated in a neuropathic pain modelOtotrauma induces sodium channel plasticity in auditory afferent neuronsDifferential expression of the capsaicin receptor TRPV1 and related novel receptors TRPV3, TRPV4 and TRPM8 in normal human tissues and changes in traumatic and diabetic neuropathyVoltage-gated sodium channel expression in mouse DRG after SNI leads to re-evaluation of projections of injured fibers.Homeostatic plasticity in hippocampal slice cultures involves changes in voltage-gated Na+ channel expressionSodium channels and pain.Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesiaTranscriptional and posttranslational plasticity and the generation of inflammatory painThe tarantula toxin β/δ-TRTX-Pre1a highlights the importance of the S1-S2 voltage-sensor region for sodium channel subtype selectivity.Voltage-gated Na+ currents in human dorsal root ganglion neurons.The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons.Extra spike formation in sensory neurons and the disruption of afferent spike patterningTetrodotoxin (TTX) as a therapeutic agent for pain.Identification of molecular pathologies sufficient to cause neuropathic excitability in primary somatosensory afferents using dynamical systems theory.Hyperalgesic agents increase a tetrodotoxin-resistant Na+ current in nociceptors.Varicella-zoster viruses associated with post-herpetic neuralgia induce sodium current density increases in the ND7-23 Nav-1.8 neuroblastoma cell line.Are voltage-gated sodium channels on the dorsal root ganglion involved in the development of neuropathic pain?The roles of sodium channels in nociception: Implications for mechanisms of painHypotonicity modulates tetrodotoxin-sensitive sodium current in trigeminal ganglion neuronsSodium channel expression in the ventral posterolateral nucleus of the thalamus after peripheral nerve injuryNerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.Properties of sodium currents in neonatal and young adult mouse superficial dorsal horn neurons.Sensory neuron-specific sodium channel SNS is abnormally expressed in the brains of mice with experimental allergic encephalomyelitis and humans with multiple sclerosisRegulatory Role of Voltage-Gated Na Channel β Subunits in Sensory NeuronsModulation of spinal cord synaptic activity by tumor necrosis factor α in a model of peripheral neuropathyAge dependence of clinical and pathological manifestations of autoimmune demyelination. Implications for multiple sclerosis.Assessment of TTX-s and TTX-r Action Potential Conduction along Neurites of NGF and GDNF Cultured Porcine DRG Somata.Control of hair cell excitability by vestibular primary sensory neuronsVirus-Mediated Knockdown of Nav1.3 in Dorsal Root Ganglia of STZ-Induced Diabetic Rats Alleviates Tactile Allodynia.NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy.Conotoxins targeting neuronal voltage-gated sodium channel subtypes: potential analgesics?Expression of voltage-gated sodium channel Nav1.3 is associated with severity of traumatic brain injury in adult ratsVirus-mediated shRNA knockdown of Na(v)1.3 in rat dorsal root ganglion attenuates nerve injury-induced neuropathic painMultiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons.
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Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on July 1994
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Type III sodium channel mRNA i ...... reexpressed following axotomy
@en
Type III sodium channel mRNA i ...... reexpressed following axotomy.
@nl
type
label
Type III sodium channel mRNA i ...... reexpressed following axotomy
@en
Type III sodium channel mRNA i ...... reexpressed following axotomy.
@nl
prefLabel
Type III sodium channel mRNA i ...... reexpressed following axotomy
@en
Type III sodium channel mRNA i ...... reexpressed following axotomy.
@nl
P2093
P2860
P356
P1476
Type III sodium channel mRNA i ...... reexpressed following axotomy
@en
P2093
P2860
P304
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10.1152/JN.1994.72.1.466
P407
P577
1994-07-01T00:00:00Z