CMTM3 presents a secreted form released via exosomes. - Wikidata - wikimedia - TriplyDB

CMTM3 presents a secreted form released via exosomes.

CMTM3 presents a secreted form released via exosomes.

http://www.wikidata.org/entity/Q37020071

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CMTM3 presents a secreted form released via exosomes.

http://www.wikidata.org/entity/Q37020071

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 April 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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CMTM3 presents a secreted form released via exosomes.

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CMTM3 presents a secreted form released via exosomes.

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CMTM3 presents a secreted form released via exosomes.

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CMTM3 presents a secreted form released via exosomes.

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CMTM3 presents a secreted form released via exosomes.

@en

CMTM3 presents a secreted form released via exosomes.

@nl

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Acta Biochimica et Biophysica Sinica

P1476

CMTM3 presents a secreted form released via exosomes.

@en

P2093

Baocai Liu

http://www.w3.org/2001/XMLSchema#string

Henan Li

http://www.w3.org/2001/XMLSchema#string

Hui Xue

http://www.w3.org/2001/XMLSchema#string

Wanchang Liu

http://www.w3.org/2001/XMLSchema#string

Wanqiong Yuan

http://www.w3.org/2001/XMLSchema#string

Weiwei Fu

http://www.w3.org/2001/XMLSchema#string

Xiaoning Mo

http://www.w3.org/2001/XMLSchema#string

Yingying Cheng

http://www.w3.org/2001/XMLSchema#string

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Caveolin-1 and MAL are located on prostasomes secreted by the prostate cancer PC-3 cell line

Membrane vesicles as conveyors of immune responses

Identification of eight genes encoding chemokine-like factor superfamily members 1-8 (CKLFSF1-8) by in silico cloning and experimental validation.

CMTM3, located at the critical tumor suppressor locus 16q22.1, is silenced by CpG methylation in carcinomas and inhibits tumor cell growth through inducing apoptosis.

The regulation of exosome secretion: a novel function of the p53 protein.

Multivesicular body morphogenesis requires phosphatidyl-inositol 3-kinase activity.

Post-Golgi membrane traffic: brefeldin A inhibits export from distal Golgi compartments to the cell surface but not recycling.

Pills of PTEN? In and out for tumor suppression.

CMTM5-v1, a four-transmembrane protein, presents a secreted form released via a vesicle-mediated secretory pathway.

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584-586

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scholarly article

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10.1093/ABBS/GMW029

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6

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48

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2016-04-28T00:00:00Z

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27125975

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4913520

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