Damage to Candida albicans hyphae and pseudohyphae by the myeloperoxidase system and oxidative products of neutrophil metabolism in vitro.
about
Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicansSevere impairment in early host defense against Candida albicans in mice deficient in myeloperoxidaseCytosolic calcium changes in individual neutrophils stimulated by opsonized and unopsonized Candida albicans hyphae.Cloning and sequencing of a Candida albicans catalase gene and effects of disruption of this gene.Candida albicans is phagocytosed, killed, and processed for antigen presentation by human dendritic cells.Enhanced killing of Candida albicans by human macrophages adherent to type 1 collagen matrices via induction of phagolysosomal fusion.Antioxidant systems in the pathogenic fungi of man and their role in virulence.T cells augment monocyte and neutrophil function in host resistance against oropharyngeal candidiasisDisparate effects of interferon-gamma and tumor necrosis factor-alpha on early neutrophil respiratory burst and fungicidal responses to Candida albicans hyphae in vitroCandida albicans escapes from mouse neutrophils.In vitro susceptibility of fungi to killing by neutrophil granulocytes discriminates between primary pathogenicity and opportunism.Nox2 modification of LDL is essential for optimal apolipoprotein B-mediated control of agr type III Staphylococcus aureus quorum-sensing.Role of granulocyte oxygen products in damage of Schistosoma mansoni eggs in vitro.Independence of neutrophil respiratory burst oxidant generation from the early cytosolic calcium response after stimulation with unopsonized Candida albicans hyphae.Redundant contribution of myeloperoxidase-dependent systems to neutrophil-mediated killing of Escherichia coli.Inactivation of Escherichia coli penicillin-binding proteins by human neutrophils.Participation of neutrophils and delayed hypersensitivity in the clearance of experimental cutaneous candidiasis in mice.Gene expression in HL60 granulocytoids and human polymorphonuclear leukocytes exposed to Candida albicans.Damage to Aspergillus fumigatus and Rhizopus oryzae hyphae by oxidative and nonoxidative microbicidal products of human neutrophils in vitroMetabolomics reveals differential levels of oral metabolites in HIV-infected patients: toward novel diagnostic targetsHost defence against disseminated Candida albicans infection and implications for antifungal immunotherapy.Macrophages in resistance to candidiasisElevated Candida antigen titers are associated with neutrophil dysfunction after injuryMacrophage colony-stimulating factor in murine candidiasis: serum and tissue levels during infection and protective effect of exogenous administration.Augmentation of GG2EE macrophage cell line-mediated anti-Candida activity by gamma interferon, tumor necrosis factor, and interleukin-1.Effects of neutrophils and in vitro oxidants on survival and phenotypic switching of Candida albicans WO-1.Stimulation of neutrophil actin polymerization and degranulation by opsonized and unopsonized Candida albicans hyphae and zymosan.Monocyte-mediated serum-independent damage to hyphal and pseudohyphal forms of Candida albicans in vitro.Antimicrobial activities of dialysate-elicited and resident human peritoneal macrophages.Killing of Aspergillus fumigatus spores and Candida albicans yeast phase by the iron-hydrogen peroxide-iodide cytotoxic system: comparison with the myeloperoxidase-hydrogen peroxide-halide system.Host-parasite relationship in opportunistic mycoses.Candidicidal activity of the human recombinant myeloperoxidase system.Early differential molecular response of a macrophage cell line to yeast and hyphal forms of Candida albicans.In vivo activation of peripheral blood polymorphonuclear neutrophils by gamma interferon results in enhanced fungal killing.Pathogenicity of morphological and auxotrophic mutants of Candida albicans in experimental infections.Susceptibility of Blastomyces dermatitidis strains to products of oxidative metabolism.Effect of dialysate fluids on phagocytosis and killing by normal neutrophils.The early transcriptional response of human granulocytes to infection with Candida albicans is not essential for killing but reflects cellular communications.Purification and some properties of myeloperoxidase and eosinophil peroxidase from human bloodAction of hypochlorous acid on the antioxidant protective enzymes superoxide dismutase, catalase and glutathione peroxidase.
P2860
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P2860
Damage to Candida albicans hyphae and pseudohyphae by the myeloperoxidase system and oxidative products of neutrophil metabolism in vitro.
description
1980 nî lūn-bûn
@nan
1980年の論文
@ja
1980年学术文章
@wuu
1980年学术文章
@zh-cn
1980年学术文章
@zh-hans
1980年学术文章
@zh-my
1980年学术文章
@zh-sg
1980年學術文章
@yue
1980年學術文章
@zh
1980年學術文章
@zh-hant
name
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@en
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@nl
type
label
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@en
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@nl
prefLabel
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@en
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@nl
P2093
P2860
P356
P1476
Damage to Candida albicans hyp ...... eutrophil metabolism in vitro.
@en
P2093
C C Haudenschild
R D Diamond
P2860
P304
P356
10.1172/JCI109958
P407
P577
1980-11-01T00:00:00Z