SETDB1 mediated FosB expression increases the cell proliferation rate during anticancer drug therapy.
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Ablation of human telomerase reverse transcriptase (hTERT) induces cellular senescence in gastric cancer through a galectin-3 dependent mechanism.Expression and characterization of transmembrane and coiled-coil domain family 3.Emerging role of SETDB1 as a therapeutic target.Bach2 represses the AP-1-driven induction of interleukin-2 gene transcription in CD4＋ T cellsLDB2 regulates the expression of DLL4 through the formation of oligomeric complexes in endothelial cells.
P2860
SETDB1 mediated FosB expression increases the cell proliferation rate during anticancer drug therapy.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 March 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
@en
SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
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type
label
SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
@en
SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
@nl
prefLabel
SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
@en
SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
@nl
P2093
P2860
P1433
P1476
SETDB1 mediated FosB expressio ...... uring anticancer drug therapy.
@en
P2093
Han-Heom Na
Hee-Jung Noh
Hyang-Min Cheong
Keun-Cheol Kim
Yoonsung Kang
P2860
P304
P356
10.5483/BMBREP.2016.49.4.031
P577
2016-03-07T00:00:00Z