Brain microglia were activated in sporadic CJD but almost unchanged in fatal familial insomnia and G114V genetic CJD.
about
Genetic and Transcriptomic Profiles of Inflammation in Neurodegenerative Diseases: Alzheimer, Parkinson, Creutzfeldt-Jakob and TauopathiesPrion pathogenesis in the absence of NLRP3/ASC inflammasomesSubtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.Proteomics analyses for the global proteins in the brain tissues of different human prion diseases.The Features of Genetic Prion Diseases Based on Chinese Surveillance Program.An In Vivo (11)C-(R)-PK11195 PET and In Vitro Pathology Study of Microglia Activation in Creutzfeldt-Jakob Disease.Re-infection of the prion from the scrapie‑infected cell line SMB-S15 in three strains of mice, CD1, C57BL/6 and Balb/c.Low activity of complement in the cerebrospinal fluid of the patients with various prion diseases.Endogenous retrovirus-K and nervous system diseases.The Role of Microglia in Prion Diseases: A Paradigm of Functional Diversity.Microglia in prion diseases.Increases of Galectin-1 and its S-nitrosylated form in the Brain Tissues of Scrapie-Infected Rodent Models and Human Prion Diseases.Remarkable increases of α1-antichymotrypsin in brain tissues of rodents during prion infection.Genetic prion disease: no role for the immune system in disease pathogenesis?An in vivo 11C-PK PET study of microglia activation in Fatal Familial Insomnia.Identification of new molecular alterations in fatal familial insomnia.
P2860
Q26767289-0A988B8B-A6ED-4F94-87E2-87B786C7BB23Q27316552-F42EEC92-0E7C-46A1-AD75-2087A0820C75Q33997514-C65D4B8A-13E5-4D91-89A0-40C6A7720339Q35325652-DB84C045-714B-4488-BEF5-9BDC92C49032Q35815746-1D184790-5060-4A93-BA85-AAB1AD48E706Q36358771-DFFAB0F3-A409-478C-8DE0-ABE43FFB8D22Q36630689-495BCE98-E0AE-4B7C-ACC7-7BE9AE392036Q36860739-E49116FD-50D3-4BFB-891A-AAE95F807AD5Q38241314-2537522A-6797-46CC-A35C-4EACB0B18A93Q39424451-D8219B0D-4F22-412C-AAD0-7C64FF3679D2Q40132256-216EAAC9-9A92-4681-B628-2DAA5FA2E67EQ40664403-6BF68951-E570-470C-8617-5748867B2D8FQ41928875-95F5C16B-DFCE-413F-BC0B-3C63570FE919Q42222309-2DD8DA85-1307-407F-AA56-80B9F2B04DEBQ47555639-52D7C153-01C6-4616-97D2-DF08CC438AF2Q47870132-0B9DE905-FB76-45EA-9882-F44C5CCE2B1F
P2860
Brain microglia were activated in sporadic CJD but almost unchanged in fatal familial insomnia and G114V genetic CJD.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on July 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@en
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@nl
type
label
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@en
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@nl
prefLabel
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@en
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@nl
P2093
P2860
P356
P1433
P1476
Brain microglia were activated ...... nsomnia and G114V genetic CJD.
@en
P2093
Baoyun Zhang
Li-Na Chen
Wu-Ling Xie
Xiao-Mei Zhang
Xiao-Ping Dong
P2860
P2888
P356
10.1186/1743-422X-10-216
P577
2013-07-01T00:00:00Z
P5875
P6179
1001444577