Pathophysiology of experimental leishmaniasis: pattern of development of metastatic disease in the susceptible host
about
Malnutrition alters the innate immune response and increases early visceralization following Leishmania donovani infection.Protective response to Leishmania major in BALB/c mice requires antigen processing in the absence of DM.Elimination of CD4+ suppressor T cells from susceptible BALB/c mice releases CD8+ T lymphocytes to mediate protective immunity against LeishmaniaLeishmania major Hsp100 is required chiefly in the mammalian stage of the parasitePolymerase chain reaction detection and inducible nitric-oxide synthase expression of Leishmania major in mice inoculated by two different routes.Immune adherence-mediated opsonophagocytosis: the mechanism of Leishmania infection.
P2860
Pathophysiology of experimental leishmaniasis: pattern of development of metastatic disease in the susceptible host
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on May 1986
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Pathophysiology of experimenta ...... isease in the susceptible host
@en
Pathophysiology of experimenta ...... sease in the susceptible host.
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type
label
Pathophysiology of experimenta ...... isease in the susceptible host
@en
Pathophysiology of experimenta ...... sease in the susceptible host.
@nl
prefLabel
Pathophysiology of experimenta ...... isease in the susceptible host
@en
Pathophysiology of experimenta ...... sease in the susceptible host.
@nl
P2860
P1476
Pathophysiology of experimenta ...... isease in the susceptible host
@en
P2093
P2860
P304
P407
P577
1986-05-01T00:00:00Z