CLI-095 decreases atherosclerosis by modulating foam cell formation in apolipoprotein E-deficient mice. - Wikidata - wikimedia - TriplyDB

CLI-095 decreases atherosclerosis by modulating foam cell formation in apolipoprotein E-deficient mice.

CLI-095 decreases atherosclerosis by modulating foam cell formation in apolipoprotein E-deficient mice.

http://www.wikidata.org/entity/Q37032384

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Atorvastatin attenuates p‑cresyl sulfate‑induced atherogenesis and plaque instability in ApoE knockout mice.DFMG reverses proliferation and migration of vascular smooth muscle cells induced by co-culture with injured vascular endothelial cells via suppression of the TLR4-mediated signaling pathway.

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CLI-095 decreases atherosclerosis by modulating foam cell formation in apolipoprotein E-deficient mice.

http://www.wikidata.org/entity/Q37032384

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 May 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

@en

CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

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type

label

CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

@en

CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

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prefLabel

CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

@en

CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

@nl

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Molecular Medicine Reports

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CLI-095 decreases atherosclero ...... olipoprotein E-deficient mice.

@en

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Hui-Qing Wan

http://www.w3.org/2001/XMLSchema#string

Peng Qu

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Xian-Jing Wei

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Xiao-Qing Wang

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Ying Zhang

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P2860

Macrophage-specific inhibition of NF-kappaB activation reduces foam-cell formation

An endothelial receptor for oxidized low-density lipoprotein

Increased atherosclerosis in LDL receptor-null mice lacking ACAT1 in macrophages

Impaired response of VLDL lipid and apoB secretion to endotoxin in the fasted rat liver

Inflammation, atherosclerosis, and coronary artery disease

Inflammation in atherosclerosis

Progress and challenges in translating the biology of atherosclerosis

Inhibitory effects of vinpocetine on the progression of atherosclerosis are mediated by Akt/NF-κB dependent mechanisms in apoE-/- mice

Humans with atherosclerosis have impaired ABCA1 cholesterol efflux and enhanced high-density lipoprotein oxidation by myeloperoxidase.

TLR4-mediated expression of Mac-1 in monocytes plays a pivotal role in monocyte adhesion to vascular endothelium.

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49-56

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scholarly article

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10.3892/MMR.2016.5233

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1

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14

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2016-05-10T00:00:00Z

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27176130

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atherosclerosis

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4918599

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