The microRNA cluster miR-17∼92 promotes TFH cell differentiation and represses subset-inappropriate gene expression. - Wikidata - wikimedia - TriplyDB

The microRNA cluster miR-17∼92 promotes TFH cell differentiation and represses subset-inappropriate gene expression.

The microRNA cluster miR-17∼92 promotes TFH cell differentiation and represses subset-inappropriate gene expression.

http://www.wikidata.org/entity/Q37038839

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Targeting microRNAs as key modulators of tumor immune response miRNAs as new molecular insights into inflammatory bowel disease: Crucial regulators in autoimmunity and inflammation MYC and the art of microRNA maintenance MicroRNA regulation of lymphocyte tolerance and autoimmunity Progress and challenge of microRNA research in immunity Targeting microRNAs for immunomodulation MicroRNA regulation of the germinal center response Follicular Helper T Cells in Systemic Lupus Erythematosus: Why Should They Be Considered as Interesting Therapeutic Targets?Regulators of Tfh Cell Differentiation miRNAs in the Pathogenesis of Systemic Lupus Erythematosus miR-17-92 cluster targets phosphatase and tensin homology and Ikaros Family Zinc Finger 4 to promote TH17-mediated inflammation Association between mir-24 and mir-378 in formalin-fixed paraffin-embedded tissues of breast cancer.Canonical microRNAs in thymic epithelial cells promote central tolerance.microRNA-17-92 regulates IL-10 production by regulatory T cells and control of experimental autoimmune encephalomyelitis.Complex interactions of transcription factors in mediating cytokine biology in T cells.Helper T cell plasticity: impact of extrinsic and intrinsic signals on transcriptomes and epigenomes.microRNA-mediated regulation of mTOR complex components facilitates discrimination between activation and anergy in CD4 T cells.Regulatory T cells in B cell follicles.Antiviral B cell and T cell immunity in the lungs.A microRNA upregulated in asthma airway T cells promotes TH2 cytokine production.The E3 ubiquitin ligase Itch is required for the differentiation of follicular helper T cells.CTLA-4 controls follicular helper T-cell differentiation by regulating the strength of CD28 engagement.MicroRNAs in hematopoietic development Chlamydia muridarum infection associated host MicroRNAs in the murine genital tract and contribution to generation of host immune response.MicroRNA-146a regulates ICOS-ICOSL signalling to limit accumulation of T follicular helper cells and germinal centres.Follicular helper T cell signature in type 1 diabetes microRNA-17-5p Modulates Bacille Calmette-Guerin Growth in RAW264.7 Cells by Targeting ULK1.Tracking early T follicular helper cell differentiation in vivo.Over-expression of miR-675 in formalin-fixed paraffin-embedded (FFPE) tissues of breast cancer patients.Follicular Helper T Cells in Autoimmunity.Functional interactions among members of the miR-17-92 cluster in lymphocyte development, differentiation and malignant transformation.miRNA92a targets KLF2 and the phosphatase PTEN signaling to promote human T follicular helper precursors in T1D islet autoimmunity.Phenotypic Tfh development promoted by CXCR5-controlled re-localization and IL-6 from radiation-resistant cells Differential Sensitivity of Target Genes to Translational Repression by miR-17~92.Transfection of microRNA Mimics Should Be Used with Caution.miR-23∼27∼24 clusters control effector T cell differentiation and function.A miR-155-Peli1-c-Rel pathway controls the generation and function of T follicular helper cells.Cutting Edge: miR-17-92 Is Required for Both CD4 Th1 and T Follicular Helper Cell Responses during Viral Infection.Critical roles of mTOR Complex 1 and 2 for T follicular helper cell differentiation and germinal center responses.Insights into the role of follicular helper T cells in autoimmunity.

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The microRNA cluster miR-17∼92 promotes TFH cell differentiation and represses subset-inappropriate gene expression.

http://www.wikidata.org/entity/Q37038839

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 30 June 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

The microRNA cluster miR-17∼92 ...... inappropriate gene expression.

@en

The microRNA cluster miR-17∼92 ...... inappropriate gene expression.

@nl

type

label

The microRNA cluster miR-17∼92 ...... inappropriate gene expression.

@en

The microRNA cluster miR-17∼92 ...... inappropriate gene expression.

@nl

prefLabel

The microRNA cluster miR-17∼92 ...... inappropriate gene expression.

@en

The microRNA cluster miR-17∼92 ...... inappropriate gene expression.

@nl

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Nature Immunology

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The microRNA cluster miR-17∼92 ...... -inappropriate gene expression

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Jeffrey A Bluestone

http://www.w3.org/2001/XMLSchema#string

Jonathan M Clingan

http://www.w3.org/2001/XMLSchema#string

Malika M Morar

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Mehrdad Matloubian

http://www.w3.org/2001/XMLSchema#string

Oliver Bannard

http://www.w3.org/2001/XMLSchema#string

Robin Kageyama

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Sana Patel

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P2860

Germinal center T follicular helper cell IL-4 production is dependent on signaling lymphocytic activation molecule receptor (CD150)

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

Lymphoproliferative disease and autoimmunity in mice with increased miR-17-92 expression in lymphocytes

miR-19 is a key oncogenic component of mir-17-92

Most mammalian mRNAs are conserved targets of microRNAs

MicroRNAs in stress signaling and human disease

Microarray analysis shows that some microRNAs downregulate large numbers of target mRNAs

Bcl6 and Blimp-1 are reciprocal and antagonistic regulators of T follicular helper cell differentiation

Bcl6 mediates the development of T follicular helper cells

Phosphoinositide 3-kinase activity in T cells regulates the magnitude of the germinal center reaction

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840-848

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scholarly article

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10.1038/NI.2642

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8

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14

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Dirk Baumjohann

Dimitri de Kouchkovsky

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2013-06-30T00:00:00Z

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243968091

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23812098

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3720769

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