Targeted deletion of SAP1 abolishes the expression of infectivity factors necessary for successful malaria parasite liver infection.
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Malaria parasite pre-erythrocytic stage infection: gliding and hidingTransition of Plasmodium sporozoites into liver stage-like forms is regulated by the RNA binding protein PumilioThe Fatty Acid Biosynthesis Enzyme FabI Plays a Key Role in the Development of Liver-Stage Malarial ParasitesIdentification of Novel Pre-Erythrocytic Malaria Antigen Candidates for Combination Vaccines with Circumsporozoite ProteinIdentification of an AP2-family protein that is critical for malaria liver stage developmentPlasmodium falciparum PF10_0164 (ETRAMP10.3) is an essential parasitophorous vacuole and exported protein in blood stagesProtective immunity to liver-stage malariaTranscriptional analysis of the pre-erythrocytic stages of the rodent malaria parasite, Plasmodium yoeliiA Plasmodium yoelii Mei2-Like RNA Binding Protein Is Essential for Completion of Liver Stage SchizogonyA Novel and Conserved Plasmodium Sporozoite Membrane Protein SPELD is Required for Maturation of Exo-erythrocytic FormsA rapid and robust selection procedure for generating drug-selectable marker-free recombinant malaria parasites.Gene disruption of Plasmodium falciparum p52 results in attenuation of malaria liver stage development in cultured primary human hepatocytes.Plasmepsin 4-deficient Plasmodium berghei are virulence attenuated and induce protective immunity against experimental malaria.Why functional pre-erythrocytic and bloodstage malaria vaccines fail: a meta-analysis of fully protective immunizations and novel immunological model.Extreme CD8 T cell requirements for anti-malarial liver-stage immunity following immunization with radiation attenuated sporozoites.Role of Plasmodium berghei cGMP-dependent protein kinase in late liver stage developmentMalaria parasite development in the mosquito and infection of the mammalian host.Engineering of Genetically Arrested Parasites (GAPs) For a Precision Malaria VaccineThe Puf-family RNA-binding protein Puf2 controls sporozoite conversion to liver stages in the malaria parasite.Advances and challenges in malaria vaccine development.The Apicomplexan AP2 family: integral factors regulating Plasmodium development.Malaria Parasite Liver Infection and Exoerythrocytic Biology.Quantitative bioluminescent imaging of pre-erythrocytic malaria parasite infection using luciferase-expressing Plasmodium yoeliiHost cell phosphatidylcholine is a key mediator of malaria parasite survival during liver stage infection.A genetically attenuated malaria vaccine candidate based on P. falciparum b9/slarp gene-deficient sporozoitesSuperior antimalarial immunity after vaccination with late liver stage-arresting genetically attenuated parasitesA monoallelic deletion of the TcCRT gene increases the attenuation of a cultured Trypanosoma cruzi strain, protecting against an in vivo virulent challenge.Mechanisms of stage-transcending protection following immunization of mice with late liver stage-arresting genetically attenuated malaria parasites.A next-generation genetically attenuated Plasmodium falciparum parasite created by triple gene deletion.A systematic analysis of the early transcribed membrane protein family throughout the life cycle of Plasmodium yoeliiTranslational Control of UIS4 Protein of the Host-Parasite Interface Is Mediated by the RNA Binding Protein Puf2 in Plasmodium berghei SporozoitesTailoring a Combination Preerythrocytic Malaria VaccinePre-erythrocytic malaria vaccines: identifying the targets.Genetically engineered, attenuated whole-cell vaccine approaches for malaria.Protective efficacy and safety of liver stage attenuated malaria parasitesDistinct malaria parasite sporozoites reveal transcriptional changes that cause differential tissue infection competence in the mosquito vector and mammalian hostTorins are potent antimalarials that block replenishment of Plasmodium liver stage parasitophorous vacuole membrane proteins.Multidrug ATP-binding cassette transporters are essential for hepatic development of Plasmodium sporozoites.Type II fatty acid synthesis is essential only for malaria parasite late liver stage developmentImmunogenicity, protective efficacy and safety of a recombinant DNA vaccine encoding truncated Plasmodium yoelii sporozoite asparagine-rich protein 1 (PySAP1).
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Targeted deletion of SAP1 abolishes the expression of infectivity factors necessary for successful malaria parasite liver infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 05 May 2008
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
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name
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
@en
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
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type
label
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
@en
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
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prefLabel
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
@en
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
@nl
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P2860
P921
P1476
Targeted deletion of SAP1 abol ...... aria parasite liver infection.
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P2093
Ahmed S I Aly
Hilda Silva Rivera
Isabelle Coppens
Mehdi Labaied
Nelly Camargo
Sebastian A Mikolajczak
Vanessa Jacobs-Lorena
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P304
P356
10.1111/J.1365-2958.2008.06271.X
P407
P577
2008-05-05T00:00:00Z