Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse. - Wikidata - wikimedia - TriplyDB

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

http://www.wikidata.org/entity/Q37049866

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Ribosomal Protein S6 Phosphorylation in the Nervous System: From Regulation to Function Glutamatergic targets for new alcohol medications Molecular and neurologic responses to chronic alcohol use Alcohol sensory processing and its relevance for ingestion Amygdala Dopamine Receptors Are Required for the Destabilization of a Reconsolidating Appetitive Memory(1,2).Re-exposure to nicotine-associated context from adolescence enhances alcohol intake in adulthood.mTORC1 inhibition in the nucleus accumbens 'protects' against the expression of drug seeking and 'relapse' and is associated with reductions in GluA1 AMPAR and CAMKIIα levels.Intermittent ethanol access schedule in rats as a preclinical model of alcohol abuse.The use of virtual reality in craving assessment and cue-exposure therapy in substance use disorders.Neurobiology of addiction: a neurocircuitry analysis.Neuroimmune pathways in alcohol consumption: evidence from behavioral and genetic studies in rodents and humans A new approach to researching the etiology of cannabis use disorder: integrating transmissible and nontransmissible risk within a developmental framework.The synaptoneurosome transcriptome: a model for profiling the emolecular effects of alcohol From circuits to behaviour in the amygdala.S6 Kinase Reflects and Regulates Ethanol-Induced Sedation.Bidirectional Modulation of Alcohol-Associated Memory Reconsolidation through Manipulation of Adrenergic Signaling.The First Alcohol Drink Triggers mTORC1-Dependent Synaptic Plasticity in Nucleus Accumbens Dopamine D1 Receptor Neurons.Translational control in synaptic plasticity and cognitive dysfunction Distinctive Roles of 5-aza-2'-deoxycytidine in Anterior Agranular Insular and Basolateral Amygdala in Reconsolidation of Aversive Memory Associated with Morphine in Rats.Functional regulation of PI3K-associated signaling in the accumbens by binge alcohol drinking in male but not female mice.Biotic acts of antibiotics Recruitment of a Neuronal Ensemble in the Central Nucleus of the Amygdala Is Required for Alcohol Dependence.mTORC1-dependent translation of collapsin response mediator protein-2 drives neuroadaptations underlying excessive alcohol-drinking behaviors.Persistent long-term facilitation at an identified synapse becomes labile with activation of short-term heterosynaptic plasticity.Label-Free Neuroproteomics of the Hippocampal-Accumbal Circuit Reveals Deficits in Neurotransmitter and Neuropeptide Signaling in Mice Lacking Ethanol-Sensitive Adenosine Transporter mTOR complex 1: a key player in neuroadaptations induced by drugs of abuse.Synaptic adaptations by alcohol and drugs of abuse: changes in microRNA expression and mRNA regulation.mTOR in Brain Physiology and Pathologies.Knockdown of zif268 in the posterior dorsolateral striatum does not enduringly disrupt a response memory of a rewarded T-maze task.Molecular mechanisms underlying alcohol-drinking behaviours Glial cell line-derived neurotrophic factor (GDNF) is an endogenous protector in the mesolimbic system against excessive alcohol consumption and relapse.Memory Reconsolidation.Inhibition of protein synthesis but not β-adrenergic receptors blocks reconsolidation of a cocaine-associated cue memory.FMRP Mediates Chronic Ethanol-Induced Changes in NMDA, Kv4.2, and KChIP3 Expression in the Hippocampus.Prosapip1-Dependent Synaptic Adaptations in the Nucleus Accumbens Drive Alcohol Intake, Seeking, and Reward.A new generation of mTORC1 inhibitor attenuates alcohol intake and reward in mice.Region specific activation of the AKT and mTORC1 pathway in response to excessive alcohol intake in rodents.The Arf6 activator Efa6/PSD3 confers regional specificity and modulates ethanol consumption in Drosophila and humans.Psychedelics and reconsolidation of traumatic and appetitive maladaptive memories: focus on cannabinoids and ketamine.VTA mTOR Signaling Regulates Dopamine Dynamics, Cocaine-Induced Synaptic Alterations, and Reward.

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Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

http://www.wikidata.org/entity/Q37049866

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 23 June 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@en

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@nl

type

label

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@en

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@nl

prefLabel

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@en

Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@nl

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Nature Neuroscience

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Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.

@en

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Dorit Ron

http://www.w3.org/2001/XMLSchema#string

Feng Liu

http://www.w3.org/2001/XMLSchema#string

Jeremie Neasta

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Patricia H Janak

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Quinn V Yowell

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Sami Ben Hamida

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Segev Barak

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Viktor Kharazia

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P2860

mTOR signaling in growth control and disease

mTOR signaling: at the crossroads of plasticity, memory and disease

GDNF is a fast-acting potent inhibitor of alcohol consumption and relapse

Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval

Cannabinoid modulation of hippocampal long-term memory is mediated by mTOR signaling

The translation repressor 4E-BP2 is critical for eIF4F complex formation, synaptic plasticity, and memory in the hippocampus

Brain-derived neurotrophic factor induces mammalian target of rapamycin-dependent local activation of translation machinery and protein synthesis in neuronal dendrites

A Memory Retrieval-Extinction Procedure to Prevent Drug Craving and Relapse

mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists

Update on neuropharmacological treatments for alcoholism: scientific basis and clinical findings.

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1111-1117

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scholarly article

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10.1038/NN.3439

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8

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2013-06-23T00:00:00Z

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241693192

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23792945

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