Colonization of the mouse intestine by an avirulent Salmonella typhimurium strain and its lipopolysaccharide-defective mutants.
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Orally administered P22 phage tailspike protein reduces salmonella colonization in chickens: prospects of a novel therapy against bacterial infectionsIncreased fidelity reduces poliovirus fitness and virulence under selective pressure in miceIdentification of novel factors involved in modulating motility of Salmonella enterica serotype typhimuriumPathogenicity of Salmonella enterica in Caenorhabditis elegans relies on disseminated oxidative stress in the infected hostSimultaneous assay of every Salmonella Typhi gene using one million transposon mutants.Infection of mice by Salmonella enterica serovar Enteritidis involves additional genes that are absent in the genome of serovar Typhimurium.Capsaicin-sensitive vagal afferent neurons contribute to the detection of pathogenic bacterial colonization in the gutAntigen 43-mediated autotransporter display, a versatile bacterial cell surface presentation systemAltering the length of the lipopolysaccharide O antigen has an impact on the interaction of Salmonella enterica serovar Typhimurium with macrophages and complement.Enhanced biofilm formation by Escherichia coli LPS mutants defective in Hep biosynthesisLipopolysaccharides belonging to different Salmonella serovars are differentially capable of activating Toll-like receptor 4.Neither motility nor chemotaxis plays a role in the ability of Escherichia coli F-18 to colonize the streptomycin-treated mouse large intestine.Porphyromonas gingivalis galE is involved in lipopolysaccharide O-antigen synthesis and biofilm formationTransepithelial signaling to neutrophils by salmonellae: a novel virulence mechanism for gastroenteritis.Role of lipopolysaccharide in colonization of the mouse intestine by Salmonella typhimurium studied by in situ hybridizationEnterobacterial common antigen mutants of Salmonella enterica serovar Typhimurium establish a persistent infection and provide protection against subsequent lethal challengeMutations at rfc or pmi attenuate Salmonella typhimurium virulence for mice.Salmonella choleraesuis strains deficient in O antigen remain fully virulent for mice by parenteral inoculation but are avirulent by oral administration.Murine antibody response to oral infection with live aroA recombinant Salmonella dublin vaccine strains expressing filamentous hemagglutinin antigen from Bordetella pertussis.Roles of motility, chemotaxis, and penetration through and growth in intestinal mucus in the ability of an avirulent strain of Salmonella typhimurium to colonize the large intestine of streptomycin-treated mice.In vivo colonization of the mouse large intestine and in vitro penetration of intestinal mucus by an avirulent smooth strain of Salmonella typhimurium and its lipopolysaccharide-deficient mutant.O-antigen-negative Salmonella enterica serovar Typhimurium is attenuated in intestinal colonization but elicits colitis in streptomycin-treated miceRegulated delayed expression of rfc enhances the immunogenicity and protective efficacy of a heterologous antigen delivered by live attenuated Salmonella enterica vaccines.Contribution of Salmonella typhimurium virulence factors to diarrheal disease in calvesKlebsiella pneumoniae capsule expression is necessary for colonization of large intestines of streptomycin-treated mice.A stochastic killing system for biological containment of Escherichia coli.Characterization and immunogenicity of EX880, a Salmonella typhi Ty21a-based clone which produces Vibrio cholerae O antigen.The outer core lipopolysaccharide of Salmonella enterica serovar Typhi is required for bacterial entry into epithelial cells.Clinical significance of translocation.Acute-phase concentrations of lipopolysaccharide (LPS)-binding protein inhibit innate immune cell activation by different LPS chemotypes via different mechanisms.The O-antigen negative ∆wbaV mutant of Salmonella enterica serovar Enteritidis shows adaptive resistance to antimicrobial peptides and elicits colitis in streptomycin pretreated mouse model.Frontline defenders: Goblet cell mediators dictate host-microbe interactions in the intestinal tract during health and disease.
P2860
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P2860
Colonization of the mouse intestine by an avirulent Salmonella typhimurium strain and its lipopolysaccharide-defective mutants.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 1985
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Colonization of the mouse inte ...... ysaccharide-defective mutants.
@en
Colonization of the mouse inte ...... ysaccharide-defective mutants.
@nl
type
label
Colonization of the mouse inte ...... ysaccharide-defective mutants.
@en
Colonization of the mouse inte ...... ysaccharide-defective mutants.
@nl
prefLabel
Colonization of the mouse inte ...... ysaccharide-defective mutants.
@en
Colonization of the mouse inte ...... ysaccharide-defective mutants.
@nl
P2093
P2860
P1476
Colonization of the mouse inte ...... lysaccharide-defective mutants
@en
P2093
P2860
P304
P407
P577
1985-10-01T00:00:00Z