Signal transduction inhibition of APCs diminishes th17 and Th1 responses in experimental autoimmune encephalomyelitis - Wikidata - wikimedia - TriplyDB

Signal transduction inhibition of APCs diminishes th17 and Th1 responses in experimental autoimmune encephalomyelitis

Signal transduction inhibition of APCs diminishes th17 and Th1 responses in experimental autoimmune encephalomyelitis

http://www.wikidata.org/entity/Q37055766

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Immune modulation by chondroitin sulfate and its degraded disaccharide product in the development of an experimental model of multiple sclerosis FLT-3 expression and function on microglia in multiple sclerosis.Therapeutic targeting of Syk in autoimmune diabetes Estrogen receptor-β ligand treatment modulates dendritic cells in the target organ during autoimmune demyelinating disease.Cutting edge: The transcription factor Kruppel-like factor 4 regulates the differentiation of Th17 cells independently of RORγt Flt3L controls the development of radiosensitive dendritic cells in the meninges and choroid plexus of the steady-state mouse brain.Disparate Effects of Mesenchymal Stem Cells in Experimental Autoimmune Encephalomyelitis and Cuprizone-Induced Demyelination.Eriocalyxin B ameliorates experimental autoimmune encephalomyelitis by suppressing Th1 and Th17 cells.Paeoniflorin Ameliorates Experimental Autoimmune Encephalomyelitis via Inhibition of Dendritic Cell Function and Th17 Cell Differentiation.Th17 cells induce Th1-polarizing monocyte-derived dendritic cells Opportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases.The role of dendritic cells in CNS autoimmunity.Role of immune responses in the pathogenesis of low-risk MDS and high-risk MDS: implications for immunotherapy.Brain dendritic cells: biology and pathology.Receptor Tyrosine Kinase and Tyrosine Kinase Inhibitors: New Hope for Success in Multiple Sclerosis Therapy Tyrosine Kinase Inhibitor as a new Therapy for Ischemic Stroke and other Neurologic Diseases: is there any Hope for a Better Outcome?Role of the JAK/STAT signaling pathway in regulation of innate immunity in neuroinflammatory diseases.Mesenchymal stem cells differentially modulate effector CD8+ T cell subsets and exacerbate experimental autoimmune encephalomyelitis.Modulation of dendritic cell properties by laquinimod as a mechanism for modulating multiple sclerosis.FTY720 (fingolimod) treatment tips the balance towards less immunogenic antigen-presenting cells in patients with multiple sclerosis.Marine-derived protein kinase inhibitors for neuroinflammatory diseases.

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Signal transduction inhibition of APCs diminishes th17 and Th1 responses in experimental autoimmune encephalomyelitis

http://www.wikidata.org/entity/Q37055766

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Signal transduction inhibition ...... l autoimmune encephalomyelitis

@en

Signal transduction inhibition ...... autoimmune encephalomyelitis.

@nl

type

label

Signal transduction inhibition ...... l autoimmune encephalomyelitis

@en

Signal transduction inhibition ...... autoimmune encephalomyelitis.

@nl

prefLabel

Signal transduction inhibition ...... l autoimmune encephalomyelitis

@en

Signal transduction inhibition ...... autoimmune encephalomyelitis.

@nl

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JIMMUNOL.0803631

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Journal of Immunology

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Signal transduction inhibition ...... l autoimmune encephalomyelitis

@en

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David Kardian

http://www.w3.org/2001/XMLSchema#string

Donald Small

http://www.w3.org/2001/XMLSchema#string

Erin McCadden

http://www.w3.org/2001/XMLSchema#string

Katharine A Whartenby

http://www.w3.org/2001/XMLSchema#string

Mario Skarica

http://www.w3.org/2001/XMLSchema#string

Tianhong Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Antiapoptotic activity of Stat5 required during terminal stages of myeloid differentiation

Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

Rac1 negatively regulates lipopolysaccharide-induced IL-23 p19 expression in human macrophages and dendritic cells and NF-kappaB p65 trans activation plays a novel role

Lestaurtinib (CEP701) is a JAK2 inhibitor that suppresses JAK2/STAT5 signaling and the proliferation of primary erythroid cells from patients with myeloproliferative disorders

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

A FLT3-targeted tyrosine kinase inhibitor is cytotoxic to leukemia cells in vitro and in vivo

Transforming growth factor-beta induces development of the T(H)17 lineage

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

The receptor tyrosine kinase Flt3 is required for dendritic cell development in peripheral lymphoid tissues

P304

4192-4199

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scholarly article

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10.4049/JIMMUNOL.0803631

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P407

P433

7

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182

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Peter A Calabresi

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2009-04-01T00:00:00Z

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24213585

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19299717

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experimental autoimmune encephalomyelitis

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3727416

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