Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients. - Wikidata - wikimedia - TriplyDB

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

http://www.wikidata.org/entity/Q37059849

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Ursodeoxycholic acid inhibits the proliferation of colon cancer cells by regulating oxidative stress and cancer stem-like cell growth.

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Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

http://www.wikidata.org/entity/Q37059849

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 23 February 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@en

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@nl

type

label

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@en

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@nl

prefLabel

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@en

Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@nl

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1940-6207.CAPR-15-0276

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Cancer Prevention Research

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Clinical Study of Ursodeoxycholic Acid in Barrett's Esophagus Patients.

@en

P2093

Bhaskar Banerjee

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Blake A Gibson

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Chiu-Hsieh Hsu

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Ellen Richmond

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Eugene Trowers

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Gary Della'Zanna

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H-H Sherry Chow

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Jessica A Martinez

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P2860

In Barrett's esophagus patients and Barrett's cell lines, ursodeoxycholic acid increases antioxidant expression and prevents DNA damage by bile acids.

Absolute quantification of somatic DNA alterations in human cancer

Bile acids as carcinogens in human gastrointestinal cancers.

Genomic catastrophes frequently arise in esophageal adenocarcinoma and drive tumorigenesis.

Deoxycholic acid causes DNA damage while inducing apoptotic resistance through NF-κB activation in benign Barrett's epithelial cells.

Metformin does not reduce markers of cell proliferation in esophageal tissues of patients with Barrett's esophagus.

Toxic bile acids in gastro-oesophageal reflux disease: influence of gastric acidity

Bile acids in combination with low pH induce oxidative stress and oxidative DNA damage: relevance to the pathogenesis of Barrett's oesophagus.

Assessment of Esophageal Adenocarcinoma Risk Using Somatic Chromosome Alterations in Longitudinal Samples in Barrett's Esophagus

Gastrointestinal adenocarcinomas of the esophagus, stomach, and colon exhibit distinct patterns of genome instability and oncogenesis.

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528-533

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scholarly article

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10.1158/1940-6207.CAPR-15-0276

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7

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9

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Nicholas J Shaheen

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2016-02-23T00:00:00Z

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26908564

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4930711

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