Addiction is a Reward Deficit and Stress Surfeit Disorder.
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Overlapping Mechanisms of Stress-Induced Relapse to Opioid Use Disorder and Chronic Pain: Clinical ImplicationsA Possible Role of Anhedonia as Common Substrate for Depression and AnxietyNeuropeptide regulation of signaling and behavior in the BNST.Allostasis as a conceptual framework linking bipolar disorder and addictionThinking after Drinking: Impaired Hippocampal-Dependent Cognition in Human Alcoholics and Animal Models of Alcohol DependenceA Trigger for Opioid Misuse: Chronic Pain and Stress Dysregulate the Mesolimbic Pathway and Kappa Opioid SystemMolecular mechanisms of synaptic remodeling in alcoholismElectrochemical Analysis of NeurotransmittersInvolvement of dopamine D2 receptors in addictive-like behaviour for acetaldehydeBrain reward and stress systems in addiction.Obsessive-compulsive aspects and pathological gambling in an Italian sample.Synthetic cathinones and their rewarding and reinforcing effects in rodents.Chronic stress may facilitate the recruitment of habit- and addiction-related neurocircuitries through neuronal restructuring of the striatum.Family history density of alcoholism relates to left nucleus accumbens volume in adolescent girls.Gene expression changes in serotonin, GABA-A receptors, neuropeptides and ion channels in the dorsal raphe nucleus of adolescent alcohol-preferring (P) rats following binge-like alcohol drinkingThe epigenetic landscape of alcoholism.Chronic wheel running-induced reduction of extinction and reinstatement of methamphetamine seeking in methamphetamine dependent rats is associated with reduced number of periaqueductal gray dopamine neurons.MicroRNA-30a-5p in the prefrontal cortex controls the transition from moderate to excessive alcohol consumption.Postsynaptic adenosine A2A receptors modulate intrinsic excitability of pyramidal cells in the rat basolateral amygdala.Kappa opioid receptor signaling in the brain: Circuitry and implications for treatment.Binge alcohol drinking elicits persistent negative affect in mice.Moderators of Varenicline Treatment Effects in a Double-Blind, Placebo-Controlled Trial for Alcohol Dependence: An Exploratory Analysis.Negative mood reverses devaluation of goal-directed drug-seeking favouring an incentive learning account of drug dependenceHyperactivation to pleasant interoceptive stimuli characterizes the transition to stimulant addiction.Change in psychiatric symptomatology after benfotiamine treatment in males is related to lifetime alcoholism severity.Increased Basolateral Amygdala Pyramidal Cell Excitability May Contribute to the Anxiogenic Phenotype Induced by Chronic Early-Life Stress.Gamble While You Gamble: Electronic Games in Ontario Charitable Gaming Centres.Gene Expression Changes in Glutamate and GABA-A Receptors, Neuropeptides, Ion Channels, and Cholesterol Synthesis in the Periaqueductal Gray Following Binge-Like Alcohol Drinking by Adolescent Alcohol-Preferring (P) Rats.Preclinical evidence implicating corticotropin-releasing factor signaling in ethanol consumption and neuroadaptation.Motivational Processes Underlying Substance Abuse Disorder.Forced swim stress increases ethanol consumption in C57BL/6J mice with a history of chronic intermittent ethanol exposureSupersensitive Kappa Opioid Receptors Promotes Ethanol Withdrawal-Related Behaviors and Reduce Dopamine Signaling in the Nucleus Accumbens.Cocaine Self-Administration Produces Long-Lasting Alterations in Dopamine Transporter Responses to CocaineStress-Induced Reinstatement of Nicotine Preference Requires Dynorphin/Kappa Opioid Activity in the Basolateral Amygdala.Stress and eating: a dual role for bombesin-like peptidesOptogenetic stimulation of VTA dopamine neurons reveals that tonic but not phasic patterns of dopamine transmission reduce ethanol self-administration.Increased Synaptic Excitation and Abnormal Dendritic Structure of Prefrontal Cortex Layer V Pyramidal Neurons following Prolonged Binge-Like Consumption of Ethanol.The lateral habenula in addiction and depression: an anatomical, synaptic and behavioral overview.Medications development for substance-use disorders: contextual influences (dis)incentivizing pharmaceutical-industry positioning.Designing modulators of 5-hydroxytryptamine signaling to treat abuse disorders.
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Addiction is a Reward Deficit and Stress Surfeit Disorder.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Addiction is a Reward Deficit and Stress Surfeit Disorder.
@en
Addiction is a Reward Deficit and Stress Surfeit Disorder.
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type
label
Addiction is a Reward Deficit and Stress Surfeit Disorder.
@en
Addiction is a Reward Deficit and Stress Surfeit Disorder.
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prefLabel
Addiction is a Reward Deficit and Stress Surfeit Disorder.
@en
Addiction is a Reward Deficit and Stress Surfeit Disorder.
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P2860
P356
P1476
Addiction is a Reward Deficit and Stress Surfeit Disorder.
@en
P2093
George F Koob
P2860
P356
10.3389/FPSYT.2013.00072
P577
2013-08-01T00:00:00Z