NOD2 plays an important role in the inflammatory responses of microglia and astrocytes to bacterial CNS pathogens.
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Mechanisms of Borrelia burgdorferi internalization and intracellular innate immune signalingDisruption of astrocyte STAT3 signaling decreases mitochondrial function and increases oxidative stress in vitro.Microglial activation by Citrobacter koseri is mediated by TLR4- and MyD88-dependent pathways.Nod2 suppresses Borrelia burgdorferi mediated murine Lyme arthritis and carditis through the induction of tolerance.NOD2 mediates inflammatory responses of primary murine glia to Streptococcus pneumoniaePolymorphisms in Toll-like receptors 2, 4, and 9 are highly associated with hearing loss in survivors of bacterial meningitis.Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential.Analysis of nucleotide-binding oligomerization domain proteins in a murine model of pneumococcal meningitis.Single nucleotide polymorphisms in pathogen recognition receptor genes are associated with susceptibility to meningococcal meningitis in a pediatric cohort.MyD88 is pivotal for immune recognition of Citrobacter koseri and astrocyte activation during CNS infectionProphylactic and therapeutic targeting of the neurokinin-1 receptor limits neuroinflammation in a murine model of pneumococcal meningitis.NLRs in immune privileged sitesInflammatory mediator release from primary rhesus microglia in response to Borrelia burgdorferi results from the activation of several receptors and pathwaysHm-MyD88 and Hm-SARM: two key regulators of the neuroimmune system and neural repair in the medicinal leechNOD2 is involved in the inflammatory response after cerebral ischemia-reperfusion injury and triggers NADPH oxidase 2-derived reactive oxygen species.The classification of microglial activation phenotypes on neurodegeneration and regeneration in Alzheimer's disease brain.Comparative study of the effect of baicalin and its natural analogs on neurons with oxygen and glucose deprivation involving innate immune reaction of TLR2/TNFα.The Lyme disease spirochete Borrelia burgdorferi induces inflammation and apoptosis in cells from dorsal root gangliaThe multifaceted responses of primary human astrocytes and brain microvascular endothelial cells to the Lyme disease spirochete, Borrelia burgdorferi.The Therapeutic Potential of Targeting Substance P/NK-1R Interactions in Inflammatory CNS DisordersAlpha beta-crystallin expression and presentation following infection with murine gammaherpesvirus 68.Immune players in the CNS: the astrocyte.Innate immunity and neuroinflammation.Neisseria meningitidis infection: who, when and where?Inflammasome Involvement in Alzheimer's Disease.Muramyldipeptide augments the actions of lipopolysaccharide in mice by stimulating macrophages to produce pro-IL-1β and by down-regulation of the suppressor of cytokine signaling 1 (SOCS1).NOD2 contributes to the inflammatory responses of primary murine microglia and astrocytes to Staphylococcus aureus.Nucleotide-binding oligomerization domain 2 (Nod2) is dispensable for the innate immune responses of macrophages against Yersinia enterocolitica.Inflammasome activation restricts Legionella pneumophila replication in primary microglial cells through flagellin detection.Temporal pattern of Toll-like receptor 9 upregulation in neurons and glial cells following cerebral ischemia reperfusion in mice.Trained innate immunity: a salient factor in the pathogenesis of neuroimmune psychiatric disorders.NOD2 promotes dopaminergic degeneration regulated by NADPH oxidase 2 in 6-hydroxydopamine model of Parkinson's diseaseNOD2 Expression in Meningitis and Its Influence on the Blood-Brain Barrier
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NOD2 plays an important role in the inflammatory responses of microglia and astrocytes to bacterial CNS pathogens.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on March 2009
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
NOD2 plays an important role i ...... es to bacterial CNS pathogens.
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NOD2 plays an important role i ...... es to bacterial CNS pathogens.
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type
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NOD2 plays an important role i ...... es to bacterial CNS pathogens.
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NOD2 plays an important role i ...... es to bacterial CNS pathogens.
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NOD2 plays an important role i ...... es to bacterial CNS pathogens.
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NOD2 plays an important role i ...... es to bacterial CNS pathogens.
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P2093
P2860
P921
P356
P1433
P1476
NOD2 plays an important role i ...... es to bacterial CNS pathogens.
@en
P2093
Amy B Young
David G Sterka
Ian Marriott
Samantha R Furr
Vinita S Chauhan
P2860
P304
P356
10.1002/GLIA.20770
P577
2009-03-01T00:00:00Z