Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
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Activation of mTOR (mechanistic target of rapamycin) in rheumatic diseases.The mTOR/p70S6K1 signaling pathway in renal fibrosis of children with immunoglobulin A nephropathy.Roles of mTOR complexes in the kidney: implications for renal disease and transplantation.Trifluoperazine Inhibits Mesangial Cell Proliferation by Arresting Cell Cycle-Dependent MechanismsReducing mTOR augments parietal epithelial cell density in a model of acute podocyte depletion and in aged kidneys.Rapamycin Enhances Repressed Autophagy and Attenuates Aggressive Progression in a Rat Model of IgA Nephropathy.
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Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 27 October 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
@en
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
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type
label
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
@en
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
@nl
prefLabel
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
@en
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
@nl
P2093
P2860
P356
P1476
Rapamycin ameliorates IgA nephropathy via cell cycle-dependent mechanisms.
@en
P2093
Jihua Tian
Rongshan Li
Xiaoshuang Zhou
Xinyan Liu
Yanhong Wang
P2860
P304
P356
10.1177/1535370214555666
P577
2014-10-27T00:00:00Z